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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
2
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pubmed:dateCreated |
2004-2-5
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pubmed:abstractText |
Three pathways control gastric acid secretion: the gastrin-enterochromaffin-like (ECL) cell axis, the vagus-parietal cell axis, and the cholecystokinin (CCK)-D cell axis. Mice lacking gastrin or both gastrin and CCK were examined to determine the role of the hormones.
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pubmed:commentsCorrections |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
|
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pubmed:status |
MEDLINE
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pubmed:month |
Feb
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pubmed:issn |
0016-5085
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
126
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
476-87
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:14762785-Animals,
pubmed-meshheading:14762785-Cholecystokinin,
pubmed-meshheading:14762785-Enterochromaffin Cells,
pubmed-meshheading:14762785-Gastric Acid,
pubmed-meshheading:14762785-Gastrins,
pubmed-meshheading:14762785-Mice,
pubmed-meshheading:14762785-Mice, Knockout,
pubmed-meshheading:14762785-Parietal Cells, Gastric,
pubmed-meshheading:14762785-Somatostatin-Secreting Cells,
pubmed-meshheading:14762785-Vagotomy
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pubmed:year |
2004
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pubmed:articleTitle |
Altered control of gastric acid secretion in gastrin-cholecystokinin double mutant mice.
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pubmed:affiliation |
Department of Cancer Research & Molecular Medicine, Norweigian University of Science and Technology, Trondheim, Norway.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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