14736542

Source:http://linkedlifedata.com/resource/pubmed/id/14736542

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0003811, umls-concept:C0011071, umls-concept:C0015127, umls-concept:C0026882, umls-concept:C0441712, umls-concept:C1314792, umls-concept:C1419864, umls-concept:C1515655
pubmed:issue	2
pubmed:dateCreated	2004-1-22
pubmed:abstractText	Mutations in the cardiac sodium channel gene SCN5A are responsible for type-3 long QT disease (LQT3). The genesis of cardiac arrhythmias in LQT3 is multifaceted, and the aim of this study was to further explore mechanisms by which SCN5A mutations lead to arrhythmogenesis in vivo.
pubmed:grant	http://linkedlifedata.com/resource/pubmed/grant/R01 66251, http://linkedlifedata.com/resource/pubmed/grant/R01 HL066251-01A2, http://linkedlifedata.com/resource/pubmed/grant/R01 HL066251-02, http://linkedlifedata.com/resource/pubmed/grant/R01 HL066251-03, http://linkedlifedata.com/resource/pubmed/grant/R01 HL066251-04
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pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/0077427
pubmed:citationSubset	IM
pubmed:chemical	http://linkedlifedata.com/resource/pubmed/chemical/Sodium Channels, http://linkedlifedata.com/resource/pubmed/chemical/sodium channel protein type 5...
pubmed:status	MEDLINE
pubmed:month	Feb
pubmed:issn	0008-6363
pubmed:author	pubmed-author:ChungMina KMK, pubmed-author:KirschGlenn EGE, pubmed-author:RosenbaumDavid SDS, pubmed-author:TchouPatrick JPJ, pubmed-author:TianXiao-LiXL, pubmed-author:Van WagonerDavid RDR, pubmed-author:WanXiaopingX, pubmed-author:WangQingQ, pubmed-author:WuLingL, pubmed-author:YongSandro LSL
pubmed:issnType	Print
pubmed:day	1
pubmed:volume	61
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	256-67
pubmed:dateRevised	2011-7-22
pubmed:meshHeading	pubmed-meshheading:14736542-Action Potentials, pubmed-meshheading:14736542-Animals, pubmed-meshheading:14736542-Death, Sudden, Cardiac, pubmed-meshheading:14736542-Electrocardiography, Ambulatory, pubmed-meshheading:14736542-Long QT Syndrome, pubmed-meshheading:14736542-Mice, pubmed-meshheading:14736542-Mice, Transgenic, pubmed-meshheading:14736542-Models, Animal, pubmed-meshheading:14736542-Mutation, pubmed-meshheading:14736542-Sodium Channels, pubmed-meshheading:14736542-Telemetry
pubmed:year	2004
pubmed:articleTitle	Mechanisms by which SCN5A mutation N1325S causes cardiac arrhythmias and sudden death in vivo.
pubmed:affiliation	Department of Molecular Cardiology, Lerner Research Institute, The Cleveland Clinic Foundation, Department of Molecular Medicine, Cleveland Clinic Lerner College of Medicine of Case Western Reserve University, Cleveland, OH 44195, USA.
pubmed:publicationType	Journal Article, Research Support, U.S. Gov't, P.H.S., Research Support, Non-U.S. Gov't