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pubmed-article:14667641pubmed:abstractTextIschemia-reperfusion (IR) injury causes myocardial dysfunction in part through intracellular calcium overload. A recently described pharmacologic compound, MCC-135 (5-methyl-2-[1-piperazinyl] benzenesulfonic acid monohydrate, Mitsubishi Pharma Corporation), alters intracellular calcium levels. This project tested the hypothesis that MCC-135 would influence regional myocardial contractility when administered at reperfusion and after a prolonged period of ischemia.lld:pubmed
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pubmed-article:14667641pubmed:pagination2054-61; discussion 2061lld:pubmed
pubmed-article:14667641pubmed:dateRevised2007-11-14lld:pubmed
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pubmed-article:14667641pubmed:articleTitleModulation of calcium transport improves myocardial contractility and enzyme profiles after prolonged ischemia-reperfusion.lld:pubmed
pubmed-article:14667641pubmed:affiliationDivision of Cardiothoracic Surgery, Medical University of South Carolina, and the Ralph H. Johnson Veteran's Association Medical Center, Charleston, South Carolina, USA.lld:pubmed
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pubmed-article:14667641pubmed:publicationTypeResearch Support, U.S. Gov't, P.H.S.lld:pubmed
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