Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
5
pubmed:dateCreated
2003-12-10
pubmed:abstractText
Oxidative stress contributes to myocardial ischemia-reperfusion injury. We hypothesized that administration of the antioxidant N-acetylcysteine would have beneficial effects on myocardial function after cardiopulmonary bypass and cardioplegic arrest.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
AIM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Nov
pubmed:issn
0022-5223
pubmed:author
pubmed:issnType
Print
pubmed:volume
126
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
1483-8
pubmed:dateRevised
2006-11-15
pubmed:meshHeading
pubmed-meshheading:14666023-Acetylcysteine, pubmed-meshheading:14666023-Animals, pubmed-meshheading:14666023-Antioxidants, pubmed-meshheading:14666023-Cardiopulmonary Bypass, pubmed-meshheading:14666023-Disease Models, Animal, pubmed-meshheading:14666023-Dogs, pubmed-meshheading:14666023-Female, pubmed-meshheading:14666023-Heart Arrest, Induced, pubmed-meshheading:14666023-Heart Function Tests, pubmed-meshheading:14666023-Male, pubmed-meshheading:14666023-Myocardial Contraction, pubmed-meshheading:14666023-Myocardial Reperfusion Injury, pubmed-meshheading:14666023-Oxidative Stress, pubmed-meshheading:14666023-Probability, pubmed-meshheading:14666023-Random Allocation, pubmed-meshheading:14666023-Reference Values, pubmed-meshheading:14666023-Risk Assessment, pubmed-meshheading:14666023-Sensitivity and Specificity, pubmed-meshheading:14666023-Ventricular Function, Left
pubmed:year
2003
pubmed:articleTitle
The antioxidant N-acetylcysteine preserves myocardial function and diminishes oxidative stress after cardioplegic arrest.
pubmed:affiliation
Center for Microvascular and Lymphatic Studies, Department of Surgery and Anesthesiology, the University of Texas-Houston Medical School, USA. ufischer2000@hotmail.com
pubmed:publicationType
Journal Article, Comparative Study, Research Support, Non-U.S. Gov't