pubmed-article:14618237 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:14618237 | lifeskim:mentions | umls-concept:C0034693 | lld:lifeskim |
pubmed-article:14618237 | lifeskim:mentions | umls-concept:C0232338 | lld:lifeskim |
pubmed-article:14618237 | lifeskim:mentions | umls-concept:C0003018 | lld:lifeskim |
pubmed-article:14618237 | lifeskim:mentions | umls-concept:C0042401 | lld:lifeskim |
pubmed-article:14618237 | lifeskim:mentions | umls-concept:C0597357 | lld:lifeskim |
pubmed-article:14618237 | lifeskim:mentions | umls-concept:C0035331 | lld:lifeskim |
pubmed-article:14618237 | lifeskim:mentions | umls-concept:C0232105 | lld:lifeskim |
pubmed-article:14618237 | lifeskim:mentions | umls-concept:C0680242 | lld:lifeskim |
pubmed-article:14618237 | lifeskim:mentions | umls-concept:C1882115 | lld:lifeskim |
pubmed-article:14618237 | pubmed:issue | 1 | lld:pubmed |
pubmed-article:14618237 | pubmed:dateCreated | 2004-1-16 | lld:pubmed |
pubmed-article:14618237 | pubmed:abstractText | The renin angiotensin system is emerging as a potential therapeutic target for diabetic retinopathy. This study examines the effects of angiotensin-converting-enzyme inhibition by captopril and angiotensin AT(1) receptor antagonism using candesartan-cilexetil on retinal blood flow and acetylcholine-stimulated vasodilatation in normotensive diabetic rats. | lld:pubmed |
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pubmed-article:14618237 | pubmed:language | eng | lld:pubmed |
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pubmed-article:14618237 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:14618237 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:14618237 | pubmed:month | Jan | lld:pubmed |
pubmed-article:14618237 | pubmed:issn | 0012-186X | lld:pubmed |
pubmed-article:14618237 | pubmed:author | pubmed-author:HoriiTT | lld:pubmed |
pubmed-article:14618237 | pubmed:author | pubmed-author:AbikoTT | lld:pubmed |
pubmed-article:14618237 | pubmed:author | pubmed-author:FeenerE PEP | lld:pubmed |
pubmed-article:14618237 | pubmed:author | pubmed-author:ClermontA CAC | lld:pubmed |
pubmed-article:14618237 | pubmed:author | pubmed-author:AbikoAA | lld:pubmed |
pubmed-article:14618237 | pubmed:author | pubmed-author:ShoelsonB DBD | lld:pubmed |
pubmed-article:14618237 | pubmed:author | pubmed-author:BursellS-ESE | lld:pubmed |
pubmed-article:14618237 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:14618237 | pubmed:volume | 47 | lld:pubmed |
pubmed-article:14618237 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:14618237 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:14618237 | pubmed:pagination | 113-23 | lld:pubmed |
pubmed-article:14618237 | pubmed:dateRevised | 2007-11-15 | lld:pubmed |
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pubmed-article:14618237 | pubmed:year | 2004 | lld:pubmed |
pubmed-article:14618237 | pubmed:articleTitle | Angiotensin AT(1) receptor antagonism normalizes retinal blood flow and acetylcholine-induced vasodilatation in normotensive diabetic rats. | lld:pubmed |
pubmed-article:14618237 | pubmed:affiliation | Research Division and Beetham Eye Institute, Joslin Diabetes Center and Harvard Medical School, One Joslin Place, Boston, USA. | lld:pubmed |
pubmed-article:14618237 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:14618237 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
pubmed-article:14618237 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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