Linked Life Data

14615386

Source:http://linkedlifedata.com/resource/pubmed/id/14615386

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
1
pubmed:dateCreated
2004-1-6
pubmed:abstractText
Human obesity is associated with elevated leptin levels and a high risk of death from cardiovascular disease. In the present study, we investigated the effects of leptin on vascular wound healing and arterial lesion growth in mice.
pubmed:grant
pubmed:commentsCorrections
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Jan
pubmed:issn
1524-4636
pubmed:author
pubmed:issnType
Electronic
pubmed:volume
24
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
112-7
pubmed:dateRevised
2010-11-18
pubmed:meshHeading
pubmed-meshheading:14615386-Animals, pubmed-meshheading:14615386-Aorta, pubmed-meshheading:14615386-Arteriosclerosis, pubmed-meshheading:14615386-Carotid Arteries, pubmed-meshheading:14615386-Chlorides, pubmed-meshheading:14615386-Diet, Atherogenic, pubmed-meshheading:14615386-Dietary Fats, pubmed-meshheading:14615386-Disease Models, Animal, pubmed-meshheading:14615386-Ferric Compounds, pubmed-meshheading:14615386-Gene Expression Regulation, pubmed-meshheading:14615386-Hyperplasia, pubmed-meshheading:14615386-Leptin, pubmed-meshheading:14615386-Mice, pubmed-meshheading:14615386-Mice, Inbred C57BL, pubmed-meshheading:14615386-Muscle, Smooth, Vascular, pubmed-meshheading:14615386-Obesity, pubmed-meshheading:14615386-RNA, Messenger, pubmed-meshheading:14615386-Receptors, Cell Surface, pubmed-meshheading:14615386-Receptors, Leptin, pubmed-meshheading:14615386-Recombinant Proteins, pubmed-meshheading:14615386-Tunica Intima
pubmed:year
2004
pubmed:articleTitle
Leptin promotes vascular remodeling and neointimal growth in mice.
pubmed:affiliation
Department of Cardiology and Pulmonary Medicine, University of Goettingen, Goettingen, Germany.
pubmed:publicationType
Journal Article, Research Support, U.S. Gov't, P.H.S., Research Support, Non-U.S. Gov't