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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
8
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pubmed:dateCreated |
2003-11-10
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pubmed:abstractText |
Toluene diisocyanate (TDI)-induced asthma is a common cause of occupational lung disease. In addition, a sore throat is one of the complaints of TDI-exposed workers. The aim of the present study was to determine whether TDI exposure induces laryngeal and/or tracheal lesions in experimental animals.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Oct
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pubmed:issn |
0001-6489
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pubmed:author |
|
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pubmed:issnType |
Print
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pubmed:volume |
123
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
965-71
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pubmed:meshHeading |
pubmed-meshheading:14606601-Animals,
pubmed-meshheading:14606601-Antioxidants,
pubmed-meshheading:14606601-Ascorbic Acid,
pubmed-meshheading:14606601-Disease Models, Animal,
pubmed-meshheading:14606601-Guinea Pigs,
pubmed-meshheading:14606601-Larynx,
pubmed-meshheading:14606601-Male,
pubmed-meshheading:14606601-Pulmonary Eosinophilia,
pubmed-meshheading:14606601-Toluene 2,4-Diisocyanate,
pubmed-meshheading:14606601-Trachea,
pubmed-meshheading:14606601-alpha-Tocopherol
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pubmed:year |
2003
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pubmed:articleTitle |
Toluene diisocyanate exposure induces laryngo-tracheal eosinophilia, which can be ameliorated by supplementation with antioxidant vitamins in guinea pigs.
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pubmed:affiliation |
Department of Anatomy, Kagawa Medical University, Kagawa, Japan.
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pubmed:publicationType |
Journal Article
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