Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
21
pubmed:dateCreated
2003-11-5
pubmed:abstractText
Long-term tamoxifen treatment of breast cancer can result in tamoxifen-stimulated breast cancer, in which estrogen inhibits tumor growth after tamoxifen withdrawal. We investigated the molecular mechanism(s) of estradiol-induced tumor regression by using an in vivo model of tamoxifen-stimulated human breast cancer.
pubmed:grant
pubmed:commentsCorrections
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Nov
pubmed:issn
1460-2105
pubmed:author
pubmed:issnType
Electronic
pubmed:day
5
pubmed:volume
95
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
1597-608
pubmed:dateRevised
2009-11-19
pubmed:meshHeading
pubmed-meshheading:14600092-Animals, pubmed-meshheading:14600092-Antineoplastic Agents, Hormonal, pubmed-meshheading:14600092-Blotting, Western, pubmed-meshheading:14600092-Breast Neoplasms, pubmed-meshheading:14600092-Estradiol, pubmed-meshheading:14600092-Estrogen Receptor Modulators, pubmed-meshheading:14600092-Fas Ligand Protein, pubmed-meshheading:14600092-Female, pubmed-meshheading:14600092-Humans, pubmed-meshheading:14600092-In Situ Nick-End Labeling, pubmed-meshheading:14600092-Membrane Glycoproteins, pubmed-meshheading:14600092-Mice, pubmed-meshheading:14600092-Mice, Nude, pubmed-meshheading:14600092-Polymerase Chain Reaction, pubmed-meshheading:14600092-Receptor, erbB-2, pubmed-meshheading:14600092-Tamoxifen, pubmed-meshheading:14600092-Transforming Growth Factor alpha, pubmed-meshheading:14600092-Transplantation, Heterologous
pubmed:year
2003
pubmed:articleTitle
Paradoxical action of fulvestrant in estradiol-induced regression of tamoxifen-stimulated breast cancer.
pubmed:affiliation
Robert H. Lurie Comprehensive Cancer Center, Feinberg School of Medicine, Northwestern University, Chicago, IL 60611, USA.
pubmed:publicationType
Journal Article, Research Support, U.S. Gov't, P.H.S., Research Support, Non-U.S. Gov't