Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
17
pubmed:dateCreated
2003-10-23
pubmed:abstractText
Inflammation is linked to adverse outcomes in acute coronary syndromes. Myeloperoxidase, an abundant leukocyte enzyme, is elevated in culprit lesions that have fissured or ruptured in patients with sudden death from cardiac causes. Numerous lines of evidence suggest mechanistic links between myeloperoxidase and both inflammation and cardiovascular disease.
pubmed:grant
pubmed:commentsCorrections
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
AIM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Oct
pubmed:issn
1533-4406
pubmed:author
pubmed:copyrightInfo
Copyright 2003 Massachusetts Medical Society
pubmed:issnType
Electronic
pubmed:day
23
pubmed:volume
349
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
1595-604
pubmed:dateRevised
2007-11-15
pubmed:meshHeading
pubmed-meshheading:14573731-Aged, pubmed-meshheading:14573731-Biological Markers, pubmed-meshheading:14573731-C-Reactive Protein, pubmed-meshheading:14573731-Chest Pain, pubmed-meshheading:14573731-Coronary Artery Disease, pubmed-meshheading:14573731-Coronary Thrombosis, pubmed-meshheading:14573731-Creatine Kinase, pubmed-meshheading:14573731-Creatine Kinase, MB Form, pubmed-meshheading:14573731-Female, pubmed-meshheading:14573731-Humans, pubmed-meshheading:14573731-Isoenzymes, pubmed-meshheading:14573731-Male, pubmed-meshheading:14573731-Middle Aged, pubmed-meshheading:14573731-Myocardial Infarction, pubmed-meshheading:14573731-Myocardial Revascularization, pubmed-meshheading:14573731-Peroxidase, pubmed-meshheading:14573731-Prognosis, pubmed-meshheading:14573731-Risk, pubmed-meshheading:14573731-Troponin T
pubmed:year
2003
pubmed:articleTitle
Prognostic value of myeloperoxidase in patients with chest pain.
pubmed:affiliation
Department of Cell Biology, Cleveland Clinic Foundation, Cleveland 44195, USA.
pubmed:publicationType
Journal Article, Research Support, U.S. Gov't, P.H.S.