| pubmed-article:14563312 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:14563312 | lifeskim:mentions | umls-concept:C0335038 | lld:lifeskim |
| pubmed-article:14563312 | lifeskim:mentions | umls-concept:C0017355 | lld:lifeskim |
| pubmed-article:14563312 | lifeskim:mentions | umls-concept:C0567416 | lld:lifeskim |
| pubmed-article:14563312 | lifeskim:mentions | umls-concept:C0150788 | lld:lifeskim |
| pubmed-article:14563312 | lifeskim:mentions | umls-concept:C1516373 | lld:lifeskim |
| pubmed-article:14563312 | lifeskim:mentions | umls-concept:C0456387 | lld:lifeskim |
| pubmed-article:14563312 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:14563312 | pubmed:dateCreated | 2003-10-17 | lld:pubmed |
| pubmed-article:14563312 | pubmed:abstractText | The evolutionarily ancient intracellular stress response protects cells from the effects of external and internal forces which perturb cellular metabolism. Members of the major histocompatibility complex (MHC) class I-like superfamily act as cell surface indicators of the intracellular stress response. Cellular immunity employs these indicators as a cue for elimination of damaged, infected, and malignant cells, promoting the health of the individual and the evolutionary success of the species. | lld:pubmed |
| pubmed-article:14563312 | pubmed:language | eng | lld:pubmed |
| pubmed-article:14563312 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:14563312 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:14563312 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:14563312 | pubmed:month | Oct | lld:pubmed |
| pubmed-article:14563312 | pubmed:issn | 1074-7613 | lld:pubmed |
| pubmed-article:14563312 | pubmed:author | pubmed-author:ParhamPeterP | lld:pubmed |
| pubmed-article:14563312 | pubmed:author | pubmed-author:GleimerMichae... | lld:pubmed |
| pubmed-article:14563312 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:14563312 | pubmed:volume | 19 | lld:pubmed |
| pubmed-article:14563312 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:14563312 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:14563312 | pubmed:pagination | 469-77 | lld:pubmed |
| pubmed-article:14563312 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
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| pubmed-article:14563312 | pubmed:meshHeading | pubmed-meshheading:14563312... | lld:pubmed |
| pubmed-article:14563312 | pubmed:year | 2003 | lld:pubmed |
| pubmed-article:14563312 | pubmed:articleTitle | Stress management: MHC class I and class I-like molecules as reporters of cellular stress. | lld:pubmed |
| pubmed-article:14563312 | pubmed:affiliation | Program in Immunology, Stanford University School of Medicine, Stanford, CA 94305, USA. | lld:pubmed |
| pubmed-article:14563312 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:14563312 | pubmed:publicationType | Review | lld:pubmed |
| pubmed-article:14563312 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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