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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
6
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pubmed:dateCreated |
2003-11-18
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pubmed:abstractText |
Methylglyoxal (MGO) in a heat-sterilized conventional PD solution may damage peritoneal cells directly and/or indirectly by producing advanced glycation endproducts (AGEs). This study was conducted to (a) examine the acute effect of MGO on the peritoneum (including AGE formation) and (b) study the possible AGE suppressive effect of an anti-oxidant, sodium sulfite.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:issn |
0250-8095
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pubmed:author |
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pubmed:copyrightInfo |
Copyright 2003 S. Karger AG, Basel
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pubmed:issnType |
Print
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pubmed:volume |
23
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
390-4
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pubmed:dateRevised |
2007-2-14
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pubmed:meshHeading |
pubmed-meshheading:14551463-Animals,
pubmed-meshheading:14551463-Antibodies, Monoclonal,
pubmed-meshheading:14551463-Antioxidants,
pubmed-meshheading:14551463-Glycosylation End Products, Advanced,
pubmed-meshheading:14551463-Immunohistochemistry,
pubmed-meshheading:14551463-Neovascularization, Pathologic,
pubmed-meshheading:14551463-Peritoneum,
pubmed-meshheading:14551463-Pyruvaldehyde,
pubmed-meshheading:14551463-Rats,
pubmed-meshheading:14551463-Serum Albumin,
pubmed-meshheading:14551463-Sulfites
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pubmed:articleTitle |
Hyper-vascular change and formation of advanced glycation endproducts in the peritoneum caused by methylglyoxal and the effect of an anti-oxidant, sodium sulfite.
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pubmed:affiliation |
Division of Kidney and Hypertension, Tokyo Jikei University School of Medicine, Tokyo, Japan. mnakayama@jikei.ac.jp
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pubmed:publicationType |
Journal Article,
Comparative Study
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