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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
6
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pubmed:dateCreated |
1993-1-7
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pubmed:abstractText |
Acute increases in afterload and in left ventricular asynchrony both independently prolong left ventricular isovolumetric relaxation. The aim of the study was to investigate whether an increased left ventricular afterload augments left ventricular asynchrony, which in turn could mediate the afterload dependent prolongation of left ventricular isovolumetric relaxation.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:status |
MEDLINE
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pubmed:month |
Jun
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pubmed:issn |
0008-6363
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pubmed:author |
|
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pubmed:issnType |
Print
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pubmed:volume |
26
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
631-7
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pubmed:dateRevised |
2003-11-14
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pubmed:meshHeading |
pubmed-meshheading:1451143-Animals,
pubmed-meshheading:1451143-Aorta,
pubmed-meshheading:1451143-Aorta, Thoracic,
pubmed-meshheading:1451143-Constriction,
pubmed-meshheading:1451143-Dogs,
pubmed-meshheading:1451143-Female,
pubmed-meshheading:1451143-Male,
pubmed-meshheading:1451143-Myocardial Contraction,
pubmed-meshheading:1451143-Pressure,
pubmed-meshheading:1451143-Regression Analysis,
pubmed-meshheading:1451143-Systole,
pubmed-meshheading:1451143-Time Factors,
pubmed-meshheading:1451143-Ventricular Function, Left
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pubmed:year |
1992
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pubmed:articleTitle |
Afterload dependent prolongation of left ventricular relaxation: importance of asynchrony.
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pubmed:affiliation |
Abt für Herz- und Kreislaufphysiologie, Heinrich-Heine-Universität, Düsseldorf, Germany.
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pubmed:publicationType |
Journal Article
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