pubmed-article:1413526 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:1413526 | lifeskim:mentions | umls-concept:C0021311 | lld:lifeskim |
pubmed-article:1413526 | lifeskim:mentions | umls-concept:C2806455 | lld:lifeskim |
pubmed-article:1413526 | lifeskim:mentions | umls-concept:C0007634 | lld:lifeskim |
pubmed-article:1413526 | pubmed:issue | 1 | lld:pubmed |
pubmed-article:1413526 | pubmed:dateCreated | 1992-11-16 | lld:pubmed |
pubmed-article:1413526 | pubmed:abstractText | The cellular receptors for a coronavirus, mouse hepatitis virus (MHV), have been recently identified as one or more members of the carcinoembryonic antigen (CEA) family. The neurotropic JHM strain of MHV (MHV-JHM) possesses a highly fusogenic surface (S) glycoprotein. This protein is now shown to promote the spread of MHV into cells lacking the specific CEA-related MHV receptor. Resistant cells are recruited into MHV-induced syncytium with consequent production of progeny virus. Cell-to-cell spread of virus via membrane fusion without the requirement for specific cell surface receptor offers a novel way for virus to spread within infected hosts. | lld:pubmed |
pubmed-article:1413526 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:1413526 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:1413526 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:1413526 | pubmed:language | eng | lld:pubmed |
pubmed-article:1413526 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:1413526 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:1413526 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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pubmed-article:1413526 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:1413526 | pubmed:month | Nov | lld:pubmed |
pubmed-article:1413526 | pubmed:issn | 0042-6822 | lld:pubmed |
pubmed-article:1413526 | pubmed:author | pubmed-author:PerlmanSS | lld:pubmed |
pubmed-article:1413526 | pubmed:author | pubmed-author:BuchmeierM... | lld:pubmed |
pubmed-article:1413526 | pubmed:author | pubmed-author:GallagherT... | lld:pubmed |
pubmed-article:1413526 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:1413526 | pubmed:volume | 191 | lld:pubmed |
pubmed-article:1413526 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:1413526 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:1413526 | pubmed:pagination | 517-22 | lld:pubmed |
pubmed-article:1413526 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
pubmed-article:1413526 | pubmed:meshHeading | pubmed-meshheading:1413526-... | lld:pubmed |
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pubmed-article:1413526 | pubmed:meshHeading | pubmed-meshheading:1413526-... | lld:pubmed |
pubmed-article:1413526 | pubmed:year | 1992 | lld:pubmed |
pubmed-article:1413526 | pubmed:articleTitle | Cell receptor-independent infection by a neurotropic murine coronavirus. | lld:pubmed |
pubmed-article:1413526 | pubmed:affiliation | Department of Neuropharmacology, Scripps Research Institute, La Jolla, California 92037. | lld:pubmed |
pubmed-article:1413526 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:1413526 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
pubmed-article:1413526 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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