| pubmed-article:1330028 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:1330028 | lifeskim:mentions | umls-concept:C0033204 | lld:lifeskim |
| pubmed-article:1330028 | lifeskim:mentions | umls-concept:C0596235 | lld:lifeskim |
| pubmed-article:1330028 | lifeskim:mentions | umls-concept:C0936012 | lld:lifeskim |
| pubmed-article:1330028 | lifeskim:mentions | umls-concept:C0439799 | lld:lifeskim |
| pubmed-article:1330028 | lifeskim:mentions | umls-concept:C1554088 | lld:lifeskim |
| pubmed-article:1330028 | lifeskim:mentions | umls-concept:C1701901 | lld:lifeskim |
| pubmed-article:1330028 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:1330028 | pubmed:dateCreated | 1992-12-9 | lld:pubmed |
| pubmed-article:1330028 | pubmed:abstractText | The domain model of Ca2+ inactivation of Ca2+ channels, which has been used to explain rapid inactivation of whole cell Ca2+ currents in pancreatic beta cells, is applied to single-time and conditional open probability measurements on guinea pig ventricular myocyte Ca2+ channels. These two measurements greatly constrain the choice of kinetic constants in the model. Calculations with the model provide a simple quantitative explanation of recent experimental results, including a slow increase in the inactivation rate. | lld:pubmed |
| pubmed-article:1330028 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1330028 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1330028 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1330028 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1330028 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1330028 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1330028 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1330028 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1330028 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1330028 | pubmed:language | eng | lld:pubmed |
| pubmed-article:1330028 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1330028 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:1330028 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1330028 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1330028 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:1330028 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:1330028 | pubmed:issn | 0006-3495 | lld:pubmed |
| pubmed-article:1330028 | pubmed:author | pubmed-author:KeizerJJ | lld:pubmed |
| pubmed-article:1330028 | pubmed:author | pubmed-author:MarkL PLP | lld:pubmed |
| pubmed-article:1330028 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:1330028 | pubmed:volume | 63 | lld:pubmed |
| pubmed-article:1330028 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:1330028 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:1330028 | pubmed:pagination | 291-5 | lld:pubmed |
| pubmed-article:1330028 | pubmed:dateRevised | 2010-9-7 | lld:pubmed |
| pubmed-article:1330028 | pubmed:meshHeading | pubmed-meshheading:1330028-... | lld:pubmed |
| pubmed-article:1330028 | pubmed:meshHeading | pubmed-meshheading:1330028-... | lld:pubmed |
| pubmed-article:1330028 | pubmed:meshHeading | pubmed-meshheading:1330028-... | lld:pubmed |
| pubmed-article:1330028 | pubmed:meshHeading | pubmed-meshheading:1330028-... | lld:pubmed |
| pubmed-article:1330028 | pubmed:meshHeading | pubmed-meshheading:1330028-... | lld:pubmed |
| pubmed-article:1330028 | pubmed:meshHeading | pubmed-meshheading:1330028-... | lld:pubmed |
| pubmed-article:1330028 | pubmed:meshHeading | pubmed-meshheading:1330028-... | lld:pubmed |
| pubmed-article:1330028 | pubmed:meshHeading | pubmed-meshheading:1330028-... | lld:pubmed |
| pubmed-article:1330028 | pubmed:meshHeading | pubmed-meshheading:1330028-... | lld:pubmed |
| pubmed-article:1330028 | pubmed:meshHeading | pubmed-meshheading:1330028-... | lld:pubmed |
| pubmed-article:1330028 | pubmed:year | 1992 | lld:pubmed |
| pubmed-article:1330028 | pubmed:articleTitle | Conditional probability analysis for a domain model of Ca(2+)-inactivation of Ca2+ channels. | lld:pubmed |
| pubmed-article:1330028 | pubmed:affiliation | Institute of Theoretical Dynamics, University of California, Davis 95616. | lld:pubmed |
| pubmed-article:1330028 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:1330028 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
| pubmed-article:1330028 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:1330028 | lld:pubmed |
