| pubmed-article:1315783 | rdf:type | pubmed:Citation | lld:pubmed |
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| pubmed-article:1315783 | lifeskim:mentions | umls-concept:C0006556 | lld:lifeskim |
| pubmed-article:1315783 | lifeskim:mentions | umls-concept:C0062534 | lld:lifeskim |
| pubmed-article:1315783 | lifeskim:mentions | umls-concept:C1510470 | lld:lifeskim |
| pubmed-article:1315783 | lifeskim:mentions | umls-concept:C0017262 | lld:lifeskim |
| pubmed-article:1315783 | lifeskim:mentions | umls-concept:C1515979 | lld:lifeskim |
| pubmed-article:1315783 | lifeskim:mentions | umls-concept:C2911684 | lld:lifeskim |
| pubmed-article:1315783 | lifeskim:mentions | umls-concept:C1447107 | lld:lifeskim |
| pubmed-article:1315783 | lifeskim:mentions | umls-concept:C0185117 | lld:lifeskim |
| pubmed-article:1315783 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:1315783 | pubmed:dateCreated | 1992-6-11 | lld:pubmed |
| pubmed-article:1315783 | pubmed:abstractText | The addition of exogenous hepatocyte growth factor (HGF)/scatter factor (SF) to MDCK epithelial cells results in fibroblastic morphology and cell motility. We generated HGF/SF producing MDCK cells by transfection with an expression plasmid containing human HGF/SF cDNA. Production of HGF/SF by these cells induced a change from an epithelial to a fibroblastic morphology and increased cell motility. In addition, the HGF/SF producing cells acquired efficient anchorage-independent growth in soft agar but did not form tumors in nude mice. The morphological change and the stimulation of the anchorage-independent growth were prevented by anti-HGF/SF antibody, suggesting that the factor is secreted and then exerts its effects through cell surface receptors. | lld:pubmed |
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| pubmed-article:1315783 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:1315783 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:1315783 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:1315783 | pubmed:month | May | lld:pubmed |
| pubmed-article:1315783 | pubmed:issn | 0021-9525 | lld:pubmed |
| pubmed-article:1315783 | pubmed:author | pubmed-author:UeharaYY | lld:pubmed |
| pubmed-article:1315783 | pubmed:author | pubmed-author:KitamuraNN | lld:pubmed |
| pubmed-article:1315783 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:1315783 | pubmed:volume | 117 | lld:pubmed |
| pubmed-article:1315783 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:1315783 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:1315783 | pubmed:pagination | 889-94 | lld:pubmed |
| pubmed-article:1315783 | pubmed:dateRevised | 2010-9-7 | lld:pubmed |
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| pubmed-article:1315783 | pubmed:year | 1992 | lld:pubmed |
| pubmed-article:1315783 | pubmed:articleTitle | Expression of a human hepatocyte growth factor/scatter factor cDNA in MDCK epithelial cells influences cell morphology, motility, and anchorage-independent growth. | lld:pubmed |
| pubmed-article:1315783 | pubmed:affiliation | Institute for Liver Research, Kansai Medical University, Osaka, Japan. | lld:pubmed |
| pubmed-article:1315783 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:1315783 | pubmed:publicationType | In Vitro | lld:pubmed |
| pubmed-article:1315783 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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