Linked Life Data

12970158

Source:http://linkedlifedata.com/resource/pubmed/id/12970158

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
12
pubmed:dateCreated
2003-12-3
pubmed:abstractText
Obesity is the result of an imbalance between energy intake and energy expenditure. Under most circumstances, the increased availability of nutrients is tightly coupled with nutrient-sensing mechanisms that in turn activate appropriate behavioral and metabolic responses. The latter responses include decreases in food intake and the production of endogenous nutrients and increased expenditure of energy. The availability of nutrients can be sensed at central sites (mostly in the hypothalamus) or directly in peripheral tissues such as skeletal muscle and fat. The hypothalamus links the sensing of nutrients to the control of metabolism and feeding behavior. Here, we discuss how two central and peripheral nutrient-sensing mechanisms participate in this complex feedback system.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
AIM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Dec
pubmed:issn
0013-7227
pubmed:author
pubmed:issnType
Print
pubmed:volume
144
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
5172-8
pubmed:dateRevised
2011-11-17
pubmed:meshHeading
pubmed:year
2003
pubmed:articleTitle
Minireview: nutrient sensing and the regulation of insulin action and energy balance.
pubmed:affiliation
Albert Einstein College of Medicine, 1300 Morris Park Avenue, Belfer 701, Bronx, New York 10461, USA.
pubmed:publicationType
Journal Article, Review