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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
4
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pubmed:dateCreated |
2003-9-12
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pubmed:abstractText |
p-cresol, which is extensively metabolized into p-cresylglucuronide in the rat, is related to several biochemical and physiologic alterations in uremia and is not removed adequately by current hemodialysis strategies. The knowledge of its in vivo kinetic behavior could be helpful to improve the current removal strategies.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Oct
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pubmed:issn |
0085-2538
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pubmed:author |
|
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pubmed:issnType |
Print
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pubmed:volume |
64
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1365-73
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:12969155-Animals,
pubmed-meshheading:12969155-Creatinine,
pubmed-meshheading:12969155-Cresols,
pubmed-meshheading:12969155-Glucuronides,
pubmed-meshheading:12969155-Hydrolysis,
pubmed-meshheading:12969155-Male,
pubmed-meshheading:12969155-Osmolar Concentration,
pubmed-meshheading:12969155-Rats,
pubmed-meshheading:12969155-Rats, Inbred Strains,
pubmed-meshheading:12969155-Time Factors,
pubmed-meshheading:12969155-Uremia
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pubmed:year |
2003
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pubmed:articleTitle |
Comparative kinetics of the uremic toxin p-cresol versus creatinine in rats with and without renal failure.
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pubmed:affiliation |
Department of Pharmacology, Heymans Institute, University Hospital, Gent, Belgium. Gerri.lesaffer@kahosl.be
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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