| pubmed-article:12963636 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:12963636 | lifeskim:mentions | umls-concept:C0018801 | lld:lifeskim |
| pubmed-article:12963636 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
| pubmed-article:12963636 | lifeskim:mentions | umls-concept:C0031621 | lld:lifeskim |
| pubmed-article:12963636 | pubmed:issue | 17 | lld:pubmed |
| pubmed-article:12963636 | pubmed:dateCreated | 2003-10-28 | lld:pubmed |
| pubmed-article:12963636 | pubmed:abstractText | We have recently shown that genetic inactivation of phosphoinositide 3-kinase gamma (PI3Kgamma), the isoform linked to G-protein-coupled receptors, results in increased cardiac contractility with no effect on basal cell size. Signaling via the G-protein-coupled beta-adrenergic receptors has been implicated in cardiac hypertrophy and heart failure, suggesting that PI3Kgamma might play a role in the pathogenesis of heart disease. | lld:pubmed |
| pubmed-article:12963636 | pubmed:language | eng | lld:pubmed |
| pubmed-article:12963636 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:12963636 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:12963636 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:12963636 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:12963636 | pubmed:month | Oct | lld:pubmed |
| pubmed-article:12963636 | pubmed:issn | 1524-4539 | lld:pubmed |
| pubmed-article:12963636 | pubmed:author | pubmed-author:WadaTeijiT | lld:pubmed |
| pubmed-article:12963636 | pubmed:author | pubmed-author:PenningerJose... | lld:pubmed |
| pubmed-article:12963636 | pubmed:author | pubmed-author:SteinbergSusa... | lld:pubmed |
| pubmed-article:12963636 | pubmed:author | pubmed-author:ErikssonUrsU | lld:pubmed |
| pubmed-article:12963636 | pubmed:author | pubmed-author:BackxPeter... | lld:pubmed |
| pubmed-article:12963636 | pubmed:author | pubmed-author:SasakiTakehik... | lld:pubmed |
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| pubmed-article:12963636 | pubmed:author | pubmed-author:Irie-SasakiJu... | lld:pubmed |
| pubmed-article:12963636 | pubmed:author | pubmed-author:GidrewiczDomi... | lld:pubmed |
| pubmed-article:12963636 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:12963636 | pubmed:day | 28 | lld:pubmed |
| pubmed-article:12963636 | pubmed:volume | 108 | lld:pubmed |
| pubmed-article:12963636 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:12963636 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:12963636 | pubmed:pagination | 2147-52 | lld:pubmed |
| pubmed-article:12963636 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
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| pubmed-article:12963636 | pubmed:year | 2003 | lld:pubmed |
| pubmed-article:12963636 | pubmed:articleTitle | Phosphoinositide 3-kinase gamma-deficient mice are protected from isoproterenol-induced heart failure. | lld:pubmed |
| pubmed-article:12963636 | pubmed:affiliation | Department of Physiology, University Health Network, University of Toronto, 620 University Ave, Toronto, Ontario M5G 2C1, Canada. | lld:pubmed |
| pubmed-article:12963636 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:12963636 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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