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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
9
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pubmed:dateCreated |
2003-9-2
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pubmed:abstractText |
We hypothesized that the transgenic mice expressing a dominant-negative form of the CREB transcription factor (CREBA133) under the control of the cardiac myocyte-specific alpha-MHC promoter and displaying dilated cardiomyopathy would exhibit electrophysiologic abnormalities similar to those observed in human cardiomyopathy.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Sep
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pubmed:issn |
1045-3873
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
14
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
982-9
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pubmed:dateRevised |
2007-11-14
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pubmed:meshHeading |
pubmed-meshheading:12950544-Animals,
pubmed-meshheading:12950544-Cardiomyopathy, Dilated,
pubmed-meshheading:12950544-Cyclic AMP Response Element-Binding Protein,
pubmed-meshheading:12950544-Disease Models, Animal,
pubmed-meshheading:12950544-Electrocardiography,
pubmed-meshheading:12950544-Electrocardiography, Ambulatory,
pubmed-meshheading:12950544-Electrophysiologic Techniques, Cardiac,
pubmed-meshheading:12950544-Genes, Dominant,
pubmed-meshheading:12950544-Heart,
pubmed-meshheading:12950544-Mice,
pubmed-meshheading:12950544-Mice, Transgenic,
pubmed-meshheading:12950544-Transcription Factors
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pubmed:year |
2003
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pubmed:articleTitle |
Cardiac electrophysiologic abnormalities in the CREBA133 transgenic mouse model of idiopathic dilated cardiomyopathy.
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pubmed:affiliation |
Cardiac Arrhythmia Service, New England Medical Center, Boston, MA, USA.
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.
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