|
rdf:type |
|
|
lifeskim:mentions |
|
|
pubmed:issue |
9
|
|
pubmed:dateCreated |
2003-9-3
|
|
pubmed:abstractText |
Whether ventricular remodeling from hypertrophic cardiomyopathy (HCM), systemic hypertension, or other pathologies arises through a common signaling pathway or through independent molecular mechanisms is unknown. To study this, we assessed cardiac hypertrophy in a mouse model of HCM subjected to increased left ventricular (LV) load.
|
|
pubmed:keyword |
|
|
pubmed:language |
eng
|
|
pubmed:journal |
|
|
pubmed:citationSubset |
AIM
|
|
pubmed:chemical |
|
|
pubmed:status |
MEDLINE
|
|
pubmed:month |
Sep
|
|
pubmed:issn |
1524-4539
|
|
pubmed:author |
|
|
pubmed:issnType |
Electronic
|
|
pubmed:day |
2
|
|
pubmed:volume |
108
|
|
pubmed:owner |
NLM
|
|
pubmed:authorsComplete |
Y
|
|
pubmed:pagination |
1133-8
|
|
pubmed:dateRevised |
2007-11-15
|
|
pubmed:meshHeading |
pubmed-meshheading:12925456-Animals,
pubmed-meshheading:12925456-Cardiomyopathy, Hypertrophic,
pubmed-meshheading:12925456-Constriction,
pubmed-meshheading:12925456-Cyclosporine,
pubmed-meshheading:12925456-Hemodynamics,
pubmed-meshheading:12925456-Hypertrophy, Left Ventricular,
pubmed-meshheading:12925456-Male,
pubmed-meshheading:12925456-Mice,
pubmed-meshheading:12925456-Mutation,
pubmed-meshheading:12925456-Myosin Heavy Chains,
pubmed-meshheading:12925456-Pressure,
pubmed-meshheading:12925456-RNA,
pubmed-meshheading:12925456-Sarcomeres,
pubmed-meshheading:12925456-Survival Analysis
|
|
pubmed:year |
2003
|
|
pubmed:articleTitle |
Consequences of pressure overload on sarcomere protein mutation-induced hypertrophic cardiomyopathy.
|
|
pubmed:affiliation |
Department of Genetics, Harvard Medical School and Howard Hughes Medical Institute, Boston, Mass 02115, USA.
|
|
pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|
