Linked Life Data

12778802

Source:http://linkedlifedata.com/resource/pubmed/id/12778802

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
1
pubmed:dateCreated
2003-6-3
pubmed:abstractText
The detection of painful stimuli occurs primarily at the peripheral terminals of specialized sensory neurons called nociceptors. These small--diameter neurons transmit this information to the central nervous system, ultimately eliciting a perception of pain or discomfort. The capsaicin (vanilloid) receptor, an excitatory ion channel expressed at nociceptors, plays an important role in transducing thermal and inflammatory pain. Mice lacking the VR1 gene have deficits in thermal- or inflammation-induced hyperalgesia, which confirms the involvement of this channel in pain sensation, especially the sensation of heat-evoked pain.
pubmed:language
chi
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Jan
pubmed:issn
0559-7765
pubmed:author
pubmed:issnType
Print
pubmed:volume
34
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
11-5
pubmed:dateRevised
2006-11-15
pubmed:meshHeading
pubmed:year
2003
pubmed:articleTitle
[Capsaicin and its receptor--vanilloid receptor].
pubmed:affiliation
Neuroscience Research Institute, Department of Neurobiology, Peking University, Beijing 100083.
pubmed:publicationType
Journal Article, English Abstract, Review, Research Support, Non-U.S. Gov't