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rdf:type |
|
|
lifeskim:mentions |
|
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pubmed:issue |
4
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pubmed:dateCreated |
2003-4-9
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|
pubmed:abstractText |
Spinal cord ischemia has varied etiologies, and in some cases, may develop into paraplegia. This is attributable to the vulnerability of spinal motor neurons to ischemia. We evaluated the potential of the immunosuppressant cyclosporin A for treatment of spinal motor neuron damage caused by ischemia.
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pubmed:language |
eng
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|
pubmed:journal |
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pubmed:citationSubset |
AIM
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|
pubmed:chemical |
|
|
pubmed:status |
MEDLINE
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pubmed:month |
Apr
|
|
pubmed:issn |
0003-4975
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pubmed:author |
|
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pubmed:issnType |
Print
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pubmed:volume |
75
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pubmed:owner |
NLM
|
|
pubmed:authorsComplete |
Y
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pubmed:pagination |
1294-9
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:12683578-Animals,
pubmed-meshheading:12683578-Cell Death,
pubmed-meshheading:12683578-Cyclosporine,
pubmed-meshheading:12683578-Disease Models, Animal,
pubmed-meshheading:12683578-Immunosuppressive Agents,
pubmed-meshheading:12683578-Motor Neurons,
pubmed-meshheading:12683578-Paraplegia,
pubmed-meshheading:12683578-Rabbits,
pubmed-meshheading:12683578-Random Allocation,
pubmed-meshheading:12683578-Spinal Cord Ischemia
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|
pubmed:year |
2003
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|
pubmed:articleTitle |
Cyclosporin A reduces delayed motor neuron death after spinal cord ischemia in rabbits.
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|
pubmed:affiliation |
Department of Anesthesiology, Tohoku University Graduate School of Medicine, Sendai, Japan.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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