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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
12
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pubmed:dateCreated |
2003-4-1
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pubmed:abstractText |
Impaired endothelial regeneration contributes to arterial lesion formation. Endostatin is a specific inhibitor of endothelial cell growth and induces endothelial cell apoptosis. We examined the effect of endostatin overexpression on reendothelialization and neointima formation in a mouse model of arterial injury.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Apr
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pubmed:issn |
1524-4539
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pubmed:author |
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pubmed:issnType |
Electronic
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pubmed:day |
1
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pubmed:volume |
107
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1658-63
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:12668502-Adenoviridae,
pubmed-meshheading:12668502-Animals,
pubmed-meshheading:12668502-Apoptosis,
pubmed-meshheading:12668502-Arterial Occlusive Diseases,
pubmed-meshheading:12668502-Collagen,
pubmed-meshheading:12668502-Endostatins,
pubmed-meshheading:12668502-Endothelium, Vascular,
pubmed-meshheading:12668502-Gene Expression,
pubmed-meshheading:12668502-Genetic Vectors,
pubmed-meshheading:12668502-Male,
pubmed-meshheading:12668502-Mice,
pubmed-meshheading:12668502-Mice, Inbred C57BL,
pubmed-meshheading:12668502-Peptide Fragments,
pubmed-meshheading:12668502-Transgenes,
pubmed-meshheading:12668502-Wound Healing
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pubmed:year |
2003
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pubmed:articleTitle |
Decreased reendothelialization and increased neointima formation with endostatin overexpression in a mouse model of arterial injury.
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pubmed:affiliation |
Zena and Michael A. Wiener Cardiovascular Institute, Mount Sinai School of Medicine, 1425 Madison Ave, New York, NY 10029, USA.
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, Non-P.H.S.
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