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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
4
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pubmed:dateCreated |
2003-3-28
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pubmed:abstractText |
Hypoxic-ischaemic brain damage in cardiac arrest survivors is global, but postmortem histology could identify parts of the brain that are selectively vulnerable to ischaemia, comprising the thalamus and cortex. We hypothesized that hypoxic-ischaemic brain damage increases along the afferent sensory pathway with a stepwise decrease of detectable somatosensory evoked potential peaks.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:status |
MEDLINE
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pubmed:month |
Apr
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pubmed:issn |
0014-2972
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
33
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
283-7
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pubmed:dateRevised |
2004-11-17
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pubmed:meshHeading |
pubmed-meshheading:12662157-Adolescent,
pubmed-meshheading:12662157-Adult,
pubmed-meshheading:12662157-Aged,
pubmed-meshheading:12662157-Aged, 80 and over,
pubmed-meshheading:12662157-Brain Ischemia,
pubmed-meshheading:12662157-Evoked Potentials, Somatosensory,
pubmed-meshheading:12662157-Female,
pubmed-meshheading:12662157-Heart Arrest,
pubmed-meshheading:12662157-Humans,
pubmed-meshheading:12662157-Hypoxia, Brain,
pubmed-meshheading:12662157-Male,
pubmed-meshheading:12662157-Middle Aged
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pubmed:year |
2003
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pubmed:articleTitle |
Electrophysiological assessment of the afferent sensory pathway in cardiac arrest survivors.
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pubmed:affiliation |
Department of Medicine IV, University of Vienna Medical School, Austria. Edith.Bauer@akh-wien.ac.at
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pubmed:publicationType |
Journal Article
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