rdf:type |
|
lifeskim:mentions |
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pubmed:issue |
7
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pubmed:dateCreated |
2003-2-25
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pubmed:abstractText |
The progression of compensated hypertrophy to heart failure (HF) is still debated. We investigated patients with isolated valvular aortic stenosis and differing degrees of left ventricular (LV) systolic dysfunction to test the hypothesis that structural remodeling, as well as cell death, contributes to the transition to HF.
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pubmed:language |
eng
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pubmed:journal |
|
pubmed:citationSubset |
AIM
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pubmed:chemical |
|
pubmed:status |
MEDLINE
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pubmed:month |
Feb
|
pubmed:issn |
1524-4539
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pubmed:author |
|
pubmed:issnType |
Electronic
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pubmed:day |
25
|
pubmed:volume |
107
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
984-91
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pubmed:dateRevised |
2011-9-27
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pubmed:meshHeading |
pubmed-meshheading:12600911-Aged,
pubmed-meshheading:12600911-Aortic Valve Stenosis,
pubmed-meshheading:12600911-Capillaries,
pubmed-meshheading:12600911-Cardiomegaly,
pubmed-meshheading:12600911-Cell Death,
pubmed-meshheading:12600911-Cell Nucleus,
pubmed-meshheading:12600911-DNA,
pubmed-meshheading:12600911-Disease Progression,
pubmed-meshheading:12600911-Female,
pubmed-meshheading:12600911-Fibrosis,
pubmed-meshheading:12600911-Heart Failure,
pubmed-meshheading:12600911-Hemodynamics,
pubmed-meshheading:12600911-Humans,
pubmed-meshheading:12600911-Inflammation,
pubmed-meshheading:12600911-Male,
pubmed-meshheading:12600911-Models, Cardiovascular,
pubmed-meshheading:12600911-Myocytes, Cardiac,
pubmed-meshheading:12600911-Nuclear Proteins,
pubmed-meshheading:12600911-Peptidyl-Dipeptidase A,
pubmed-meshheading:12600911-Ribonucleoproteins,
pubmed-meshheading:12600911-Transforming Growth Factor beta,
pubmed-meshheading:12600911-Transforming Growth Factor beta1,
pubmed-meshheading:12600911-Ventricular Dysfunction, Left,
pubmed-meshheading:12600911-Ventricular Pressure
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pubmed:year |
2003
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pubmed:articleTitle |
Progression from compensated hypertrophy to failure in the pressure-overloaded human heart: structural deterioration and compensatory mechanisms.
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pubmed:affiliation |
Kerckhoff-Clinic, Department of Thoracic and Cardiovascular Surgery, Bad Nauheim, Germany. s.hein@kerckhoff.mpg.de
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pubmed:publicationType |
Journal Article
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