Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
7
pubmed:dateCreated
2003-2-25
pubmed:abstractText
The progression of compensated hypertrophy to heart failure (HF) is still debated. We investigated patients with isolated valvular aortic stenosis and differing degrees of left ventricular (LV) systolic dysfunction to test the hypothesis that structural remodeling, as well as cell death, contributes to the transition to HF.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
AIM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Feb
pubmed:issn
1524-4539
pubmed:author
pubmed:issnType
Electronic
pubmed:day
25
pubmed:volume
107
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
984-91
pubmed:dateRevised
2011-9-27
pubmed:meshHeading
pubmed-meshheading:12600911-Aged, pubmed-meshheading:12600911-Aortic Valve Stenosis, pubmed-meshheading:12600911-Capillaries, pubmed-meshheading:12600911-Cardiomegaly, pubmed-meshheading:12600911-Cell Death, pubmed-meshheading:12600911-Cell Nucleus, pubmed-meshheading:12600911-DNA, pubmed-meshheading:12600911-Disease Progression, pubmed-meshheading:12600911-Female, pubmed-meshheading:12600911-Fibrosis, pubmed-meshheading:12600911-Heart Failure, pubmed-meshheading:12600911-Hemodynamics, pubmed-meshheading:12600911-Humans, pubmed-meshheading:12600911-Inflammation, pubmed-meshheading:12600911-Male, pubmed-meshheading:12600911-Models, Cardiovascular, pubmed-meshheading:12600911-Myocytes, Cardiac, pubmed-meshheading:12600911-Nuclear Proteins, pubmed-meshheading:12600911-Peptidyl-Dipeptidase A, pubmed-meshheading:12600911-Ribonucleoproteins, pubmed-meshheading:12600911-Transforming Growth Factor beta, pubmed-meshheading:12600911-Transforming Growth Factor beta1, pubmed-meshheading:12600911-Ventricular Dysfunction, Left, pubmed-meshheading:12600911-Ventricular Pressure
pubmed:year
2003
pubmed:articleTitle
Progression from compensated hypertrophy to failure in the pressure-overloaded human heart: structural deterioration and compensatory mechanisms.
pubmed:affiliation
Kerckhoff-Clinic, Department of Thoracic and Cardiovascular Surgery, Bad Nauheim, Germany. s.hein@kerckhoff.mpg.de
pubmed:publicationType
Journal Article