12598081

Source:http://linkedlifedata.com/resource/pubmed/id/12598081

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0003009, umls-concept:C0016059, umls-concept:C0018787, umls-concept:C0020538, umls-concept:C0079284, umls-concept:C0205227, umls-concept:C0597357, umls-concept:C0678226, umls-concept:C1314939, umls-concept:C1412113, umls-concept:C1709305
pubmed:issue	4
pubmed:dateCreated	2003-2-24
pubmed:abstractText	We investigated if endothelin (ET)-1 and the renin-angiotensin-aldosterone system play a role in cardiac fibrosis.
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/8301365
pubmed:citationSubset	AIM
pubmed:chemical	http://linkedlifedata.com/resource/pubmed/chemical/5-(dimethylamino)-N-(3,4-dimethyl-5-..., http://linkedlifedata.com/resource/pubmed/chemical/Angiotensin II, http://linkedlifedata.com/resource/pubmed/chemical/Antihypertensive Agents, http://linkedlifedata.com/resource/pubmed/chemical/Biphenyl Compounds, http://linkedlifedata.com/resource/pubmed/chemical/Dansyl Compounds, http://linkedlifedata.com/resource/pubmed/chemical/Endothelin-1, http://linkedlifedata.com/resource/pubmed/chemical/Receptor, Angiotensin, Type 1, http://linkedlifedata.com/resource/pubmed/chemical/Receptors, Angiotensin, http://linkedlifedata.com/resource/pubmed/chemical/Receptors, Cell Surface, http://linkedlifedata.com/resource/pubmed/chemical/Sulfonamides, http://linkedlifedata.com/resource/pubmed/chemical/Tetrazoles, http://linkedlifedata.com/resource/pubmed/chemical/Vasoconstrictor Agents, http://linkedlifedata.com/resource/pubmed/chemical/bosentan, http://linkedlifedata.com/resource/pubmed/chemical/irbesartan
pubmed:status	MEDLINE
pubmed:month	Feb
pubmed:issn	0735-1097
pubmed:author	pubmed-author:BelloniAnna SAS, pubmed-author:KreutzReinholdR, pubmed-author:MartinPaulP, pubmed-author:NussdorferGastone GGG, pubmed-author:PessinaAchille CAC, pubmed-author:RossiGian PaoloGP, pubmed-author:SecciaTeresa MTM
pubmed:issnType	Print
pubmed:day	19
pubmed:volume	41
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	666-73
pubmed:dateRevised	2006-11-15
pubmed:meshHeading	pubmed-meshheading:12598081-Angiotensin II, pubmed-meshheading:12598081-Animals, pubmed-meshheading:12598081-Animals, Genetically Modified, pubmed-meshheading:12598081-Antihypertensive Agents, pubmed-meshheading:12598081-Biphenyl Compounds, pubmed-meshheading:12598081-Cardiomyopathies, pubmed-meshheading:12598081-Dansyl Compounds, pubmed-meshheading:12598081-Disease Models, Animal, pubmed-meshheading:12598081-Endothelin-1, pubmed-meshheading:12598081-Fibrosis, pubmed-meshheading:12598081-Hypertension, pubmed-meshheading:12598081-Male, pubmed-meshheading:12598081-Rats, pubmed-meshheading:12598081-Receptor, Angiotensin, Type 1, pubmed-meshheading:12598081-Receptors, Angiotensin, pubmed-meshheading:12598081-Receptors, Cell Surface, pubmed-meshheading:12598081-Sulfonamides, pubmed-meshheading:12598081-Tetrazoles, pubmed-meshheading:12598081-Time Factors, pubmed-meshheading:12598081-Vasoconstrictor Agents
pubmed:year	2003
pubmed:articleTitle	Cardiac fibrosis occurs early and involves endothelin and AT-1 receptors in hypertension due to endogenous angiotensin II.
pubmed:affiliation	Department of Clinical Methodology and Clinical-Surgical Technologies, University of Bari, Bari, Italy.
pubmed:publicationType	Journal Article, Research Support, Non-U.S. Gov't