pubmed-article:12568819 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:12568819 | lifeskim:mentions | umls-concept:C0031809 | lld:lifeskim |
pubmed-article:12568819 | lifeskim:mentions | umls-concept:C0007222 | lld:lifeskim |
pubmed-article:12568819 | lifeskim:mentions | umls-concept:C0241889 | lld:lifeskim |
pubmed-article:12568819 | lifeskim:mentions | umls-concept:C0679199 | lld:lifeskim |
pubmed-article:12568819 | lifeskim:mentions | umls-concept:C1708528 | lld:lifeskim |
pubmed-article:12568819 | pubmed:issue | 2 | lld:pubmed |
pubmed-article:12568819 | pubmed:dateCreated | 2003-2-5 | lld:pubmed |
pubmed-article:12568819 | pubmed:abstractText | Family history assessment can be used to combine population-wide health promotion and risk-reduction efforts with a high-risk, targeted approach to help reduce the burden of cardiovascular disease (CVD). Family history is an independent predictor of CVD, and the upper portion of the family history distribution explains a larger fraction of CVD in the population than can be explained by extreme values of other risk factors (e.g., blood pressure and cholesterol). A positive family history of disease captures the underlying complexities of gene-gene and gene-environment interactions by identifying families with combinations of risk factors, both measured and unmeasured, that lead to disease expression. Family history is a useful tool for identifying most prevalent cases of CVD and for population-wide disease-prevention efforts. A positive family history also identifies the relatively small subset of families in the population at highest risk for CVD who may benefit most from targeted screening and intensive intervention. | lld:pubmed |
pubmed-article:12568819 | pubmed:language | eng | lld:pubmed |
pubmed-article:12568819 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:12568819 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:12568819 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:12568819 | pubmed:month | Feb | lld:pubmed |
pubmed-article:12568819 | pubmed:issn | 0749-3797 | lld:pubmed |
pubmed-article:12568819 | pubmed:author | pubmed-author:GwinnMartaM | lld:pubmed |
pubmed-article:12568819 | pubmed:author | pubmed-author:HuntSteven... | lld:pubmed |
pubmed-article:12568819 | pubmed:author | pubmed-author:AdamsTed DTD | lld:pubmed |
pubmed-article:12568819 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:12568819 | pubmed:volume | 24 | lld:pubmed |
pubmed-article:12568819 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:12568819 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:12568819 | pubmed:pagination | 136-42 | lld:pubmed |
pubmed-article:12568819 | pubmed:dateRevised | 2004-11-17 | lld:pubmed |
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pubmed-article:12568819 | pubmed:meshHeading | pubmed-meshheading:12568819... | lld:pubmed |
pubmed-article:12568819 | pubmed:year | 2003 | lld:pubmed |
pubmed-article:12568819 | pubmed:articleTitle | Family history assessment: strategies for prevention of cardiovascular disease. | lld:pubmed |
pubmed-article:12568819 | pubmed:affiliation | Cardiovascular Genetics Research Program, Department of Internal Medicine, University of Utah School of Medicine, 410 Chipeta Way, Room 167, Salt Lake City, UT 84108, USA. steve@ucvg.med.utah.edu | lld:pubmed |
pubmed-article:12568819 | pubmed:publicationType | Journal Article | lld:pubmed |
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