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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
2
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pubmed:dateCreated |
2003-1-24
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pubmed:abstractText |
The acute administration of low-dose ethanol was demonstrated to attenuate liver injury elicited by gut ischemia/reperfusion (I/R). Nitric oxide (NO) has been found to be a modulator of adhesive interactions between leukocytes, platelets, and endothelial cells, but there has been much controversy about the effects of ethanol on NO regulation. The objective of this study was to investigate the role of NO in ethanol-reduced hepatic microvascular dysfunction elicited by gut I/R.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Feb
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pubmed:issn |
0815-9319
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
18
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
211-7
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:12542608-Alanine Transaminase,
pubmed-meshheading:12542608-Animals,
pubmed-meshheading:12542608-D-Alanine Transaminase,
pubmed-meshheading:12542608-DNA,
pubmed-meshheading:12542608-DNA Fragmentation,
pubmed-meshheading:12542608-Ethanol,
pubmed-meshheading:12542608-In Situ Nick-End Labeling,
pubmed-meshheading:12542608-Intestine, Small,
pubmed-meshheading:12542608-Ischemia,
pubmed-meshheading:12542608-Leukocytes,
pubmed-meshheading:12542608-Liver Circulation,
pubmed-meshheading:12542608-Liver Diseases,
pubmed-meshheading:12542608-Male,
pubmed-meshheading:12542608-Microcirculation,
pubmed-meshheading:12542608-Nitric Oxide,
pubmed-meshheading:12542608-Rats,
pubmed-meshheading:12542608-Rats, Wistar,
pubmed-meshheading:12542608-Reperfusion Injury
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pubmed:year |
2003
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pubmed:articleTitle |
Low-dose ethanol attenuates gut ischemia/reperfusion-induced liver injury in rats via nitric oxide production.
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pubmed:affiliation |
Department of Internal Medicine, School of Medicine, Keio University, Tokyo, Japan.
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pubmed:publicationType |
Journal Article,
Comparative Study,
Research Support, Non-U.S. Gov't
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