| pubmed-article:12534643 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:12534643 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
| pubmed-article:12534643 | lifeskim:mentions | umls-concept:C0011849 | lld:lifeskim |
| pubmed-article:12534643 | lifeskim:mentions | umls-concept:C0010763 | lld:lifeskim |
| pubmed-article:12534643 | lifeskim:mentions | umls-concept:C0441655 | lld:lifeskim |
| pubmed-article:12534643 | lifeskim:mentions | umls-concept:C0442805 | lld:lifeskim |
| pubmed-article:12534643 | lifeskim:mentions | umls-concept:C1515655 | lld:lifeskim |
| pubmed-article:12534643 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:12534643 | pubmed:dateCreated | 2003-1-21 | lld:pubmed |
| pubmed-article:12534643 | pubmed:abstractText | Cytochrome P450 2E1 (CYP2E1) is thought to activate a number of protoxins, and has been implicated in the development of liver disease. Increased hepatic expression of CYP2E1 occurs in rat models of diabetes but it is unclear whether human diabetics display a similar up-regulation. This study was designed to test the hypothesis that human diabetics experience enhanced CYP2E1 expression. | lld:pubmed |
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| pubmed-article:12534643 | pubmed:language | eng | lld:pubmed |
| pubmed-article:12534643 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:12534643 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:12534643 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:12534643 | pubmed:month | Jan | lld:pubmed |
| pubmed-article:12534643 | pubmed:issn | 0306-5251 | lld:pubmed |
| pubmed-article:12534643 | pubmed:author | pubmed-author:GorskiJ CJC | lld:pubmed |
| pubmed-article:12534643 | pubmed:author | pubmed-author:AsgharAliA | lld:pubmed |
| pubmed-article:12534643 | pubmed:author | pubmed-author:HallStephen... | lld:pubmed |
| pubmed-article:12534643 | pubmed:author | pubmed-author:MayaJuan FJF | lld:pubmed |
| pubmed-article:12534643 | pubmed:author | pubmed-author:WangZaiqiZ | lld:pubmed |
| pubmed-article:12534643 | pubmed:author | pubmed-author:LiLangL | lld:pubmed |
| pubmed-article:12534643 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:12534643 | pubmed:volume | 55 | lld:pubmed |
| pubmed-article:12534643 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:12534643 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:12534643 | pubmed:pagination | 77-85 | lld:pubmed |
| pubmed-article:12534643 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:12534643 | pubmed:meshHeading | pubmed-meshheading:12534643... | lld:pubmed |
| pubmed-article:12534643 | pubmed:year | 2003 | lld:pubmed |
| pubmed-article:12534643 | pubmed:articleTitle | Diabetes mellitus increases the in vivo activity of cytochrome P450 2E1 in humans. | lld:pubmed |
| pubmed-article:12534643 | pubmed:affiliation | Department of Medicine, Division of Clinical Pharmacology, Indiana University School of Medicine, Wishard Memorial Hospital, OPW 320, Indianapolis IN, USA. | lld:pubmed |
| pubmed-article:12534643 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:12534643 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:12534643 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| entrez-gene:25086 | entrezgene:pubmed | pubmed-article:12534643 | lld:entrezgene |
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