pubmed-article:12468805 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:12468805 | lifeskim:mentions | umls-concept:C0087111 | lld:lifeskim |
pubmed-article:12468805 | lifeskim:mentions | umls-concept:C0034721 | lld:lifeskim |
pubmed-article:12468805 | lifeskim:mentions | umls-concept:C0034693 | lld:lifeskim |
pubmed-article:12468805 | lifeskim:mentions | umls-concept:C0026336 | lld:lifeskim |
pubmed-article:12468805 | lifeskim:mentions | umls-concept:C0013604 | lld:lifeskim |
pubmed-article:12468805 | lifeskim:mentions | umls-concept:C0007784 | lld:lifeskim |
pubmed-article:12468805 | lifeskim:mentions | umls-concept:C0048470 | lld:lifeskim |
pubmed-article:12468805 | lifeskim:mentions | umls-concept:C0392756 | lld:lifeskim |
pubmed-article:12468805 | lifeskim:mentions | umls-concept:C0205421 | lld:lifeskim |
pubmed-article:12468805 | pubmed:issue | 12 | lld:pubmed |
pubmed-article:12468805 | pubmed:dateCreated | 2002-12-6 | lld:pubmed |
pubmed-article:12468805 | pubmed:abstractText | Studies indicate that thrombin plays an important role in intracerebral hemorrhage (ICH)-induced edema formation. Although thrombin is produced as the blood clots, it may be bound to fibrin and only gradually released from the clot. The time window for administration of a thrombin inhibitor to reduce ICH-induced edema is unknown. Whether this time window extends beyond the period when a thrombin inhibitor might exacerbate rebleeding is also unknown. | lld:pubmed |
pubmed-article:12468805 | pubmed:language | eng | lld:pubmed |
pubmed-article:12468805 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:12468805 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:12468805 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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pubmed-article:12468805 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:12468805 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:12468805 | pubmed:month | Dec | lld:pubmed |
pubmed-article:12468805 | pubmed:issn | 1524-4628 | lld:pubmed |
pubmed-article:12468805 | pubmed:author | pubmed-author:QiK YKY | lld:pubmed |
pubmed-article:12468805 | pubmed:author | pubmed-author:XiGuohuaG | lld:pubmed |
pubmed-article:12468805 | pubmed:author | pubmed-author:KeepRichard... | lld:pubmed |
pubmed-article:12468805 | pubmed:author | pubmed-author:HoffJulian... | lld:pubmed |
pubmed-article:12468805 | pubmed:author | pubmed-author:KitaokaTakuji... | lld:pubmed |
pubmed-article:12468805 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:12468805 | pubmed:volume | 33 | lld:pubmed |
pubmed-article:12468805 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:12468805 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:12468805 | pubmed:pagination | 3012-8 | lld:pubmed |
pubmed-article:12468805 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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pubmed-article:12468805 | pubmed:year | 2002 | lld:pubmed |
pubmed-article:12468805 | pubmed:articleTitle | Delayed argatroban treatment reduces edema in a rat model of intracerebral hemorrhage. | lld:pubmed |
pubmed-article:12468805 | pubmed:affiliation | Department of Neurosurgery, University of Michigan, Ann Arbor, USA. | lld:pubmed |
pubmed-article:12468805 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:12468805 | pubmed:publicationType | Comparative Study | lld:pubmed |
pubmed-article:12468805 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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