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rdf:type |
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lifeskim:mentions |
umls-concept:C0014597,
umls-concept:C0021853,
umls-concept:C0039194,
umls-concept:C0085358,
umls-concept:C0220905,
umls-concept:C0542341,
umls-concept:C1332717,
umls-concept:C1413244,
umls-concept:C1704675,
umls-concept:C1706438,
umls-concept:C2698600
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pubmed:issue |
5
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pubmed:dateCreated |
2002-10-29
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pubmed:abstractText |
Regulatory T cells play a role in the control of immune responses in the intestinal mucosa and their absence may predispose to inflammatory bowel disease (IBD). We have previously shown that T cells activated by intestinal epithelial cells (IECs) are suppressive in function. Our goal was to characterize the phenotype and function of T cells proliferating after interaction with IECs.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Nov
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pubmed:issn |
0016-5085
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
123
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1516-26
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pubmed:dateRevised |
2007-11-14
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pubmed:meshHeading |
pubmed-meshheading:12404227-Antigens, CD,
pubmed-meshheading:12404227-Biological Markers,
pubmed-meshheading:12404227-CD8-Positive T-Lymphocytes,
pubmed-meshheading:12404227-Cell Communication,
pubmed-meshheading:12404227-Cell Division,
pubmed-meshheading:12404227-Cells, Cultured,
pubmed-meshheading:12404227-Coculture Techniques,
pubmed-meshheading:12404227-Humans,
pubmed-meshheading:12404227-Integrin alpha Chains,
pubmed-meshheading:12404227-Intestinal Mucosa,
pubmed-meshheading:12404227-Membrane Glycoproteins,
pubmed-meshheading:12404227-Phenotype,
pubmed-meshheading:12404227-T-Lymphocyte Subsets
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pubmed:year |
2002
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pubmed:articleTitle |
Expansion of CD8+ T cells with regulatory function after interaction with intestinal epithelial cells.
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pubmed:affiliation |
Immunobiology Center, Mount Sinai School of Medicine, New York, New York 10029, USA. matthieu_allez@mssm.edu
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.,
Research Support, Non-U.S. Gov't
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