Linked Life Data

12390865

Source:http://linkedlifedata.com/resource/pubmed/id/12390865

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
5
pubmed:dateCreated
2002-10-22
pubmed:abstractText
The description of healthy individuals homozygous for the human leukocyte antigen-G (HLA-G) null allele raised doubts about the role of HLA-G in fetal-maternal tolerance. In light of recent results, we discuss this point by considering the potential activity of this null allele that might, indeed, produce functional truncated HLA-G molecules. In this context, we have recently described that, like the full-length HLA-G1, the HLA-G2, -G3, and -G4 truncated isoforms may be expressed at the cell surface and may modulate both innate and acquired immune responses.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Nov
pubmed:issn
0006-3363
pubmed:author
pubmed:issnType
Print
pubmed:volume
67
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
1375-8
pubmed:dateRevised
2011-11-17
pubmed:meshHeading
pubmed:year
2002
pubmed:articleTitle
Viewpoint on the functionality of the human leukocyte antigen-G null allele at the fetal-maternal interface.
pubmed:affiliation
Service de Recherches en Hémato-Immunologie, CEA-DSV-DRM, Hôpital St-Louis, Institut Universitaire d'Hématologie, 75475 Paris, Cedex 10, France.
pubmed:publicationType
Journal Article, Review