12383584

Source:http://linkedlifedata.com/resource/pubmed/id/12383584

Download in:

Switch to

Custom View

Named Graph Language Inference

Statements in which the resource exists as a subject.
Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0001480, umls-concept:C0020443, umls-concept:C0205227, umls-concept:C0232347, umls-concept:C0439799, umls-concept:C0441712, umls-concept:C0542341, umls-concept:C1545588, umls-concept:C1882151
pubmed:issue	7
pubmed:dateCreated	2002-10-17
pubmed:abstractText	This study aimed to clarify the role of adenosine triphosphate-sensitive K(+) (K(ATP)) channels in the no-reflow phenomenon and in its extension by hypercholesterolemia.
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/8301365
pubmed:citationSubset	AIM
pubmed:chemical	http://linkedlifedata.com/resource/pubmed/chemical/Adenosine Triphosphate, http://linkedlifedata.com/resource/pubmed/chemical/Glyburide, http://linkedlifedata.com/resource/pubmed/chemical/Nicorandil, http://linkedlifedata.com/resource/pubmed/chemical/Nitric Oxide Synthase, http://linkedlifedata.com/resource/pubmed/chemical/Potassium Channel Blockers, http://linkedlifedata.com/resource/pubmed/chemical/Potassium Channels, http://linkedlifedata.com/resource/pubmed/chemical/Vasodilator Agents, http://linkedlifedata.com/resource/pubmed/chemical/omega-N-Methylarginine
pubmed:status	MEDLINE
pubmed:month	Oct
pubmed:issn	0735-1097
pubmed:author	pubmed-author:GendaSatoshiS, pubmed-author:IchikawaYoshihikoY, pubmed-author:MikiTakayukiT, pubmed-author:MiuraTetsujiT, pubmed-author:ShimamotoKazuakiK
pubmed:issnType	Print
pubmed:day	2
pubmed:volume	40
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	1339-46
pubmed:dateRevised	2007-11-15
pubmed:meshHeading	pubmed-meshheading:12383584-Adenosine Triphosphate, pubmed-meshheading:12383584-Analysis of Variance, pubmed-meshheading:12383584-Animals, pubmed-meshheading:12383584-Chronic Disease, pubmed-meshheading:12383584-Coronary Circulation, pubmed-meshheading:12383584-Disease Models, Animal, pubmed-meshheading:12383584-Glyburide, pubmed-meshheading:12383584-Hemodynamics, pubmed-meshheading:12383584-Hypercholesterolemia, pubmed-meshheading:12383584-Male, pubmed-meshheading:12383584-Myocardial Infarction, pubmed-meshheading:12383584-Nicorandil, pubmed-meshheading:12383584-Nitric Oxide Synthase, pubmed-meshheading:12383584-Potassium Channel Blockers, pubmed-meshheading:12383584-Potassium Channels, pubmed-meshheading:12383584-Rabbits, pubmed-meshheading:12383584-Vasodilator Agents, pubmed-meshheading:12383584-omega-N-Methylarginine
pubmed:year	2002
pubmed:articleTitle	K(ATP) channel opening is an endogenous mechanism of protection against the no-reflow phenomenon but its function is compromised by hypercholesterolemia.
pubmed:affiliation	Second Department of Internal Medicine, Sapporo Medical University, Sapporo, Japan.
pubmed:publicationType	Journal Article, Research Support, Non-U.S. Gov't