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| pubmed-article:12241832 | lifeskim:mentions | umls-concept:C0155626 | lld:lifeskim |
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| pubmed-article:12241832 | lifeskim:mentions | umls-concept:C0006938 | lld:lifeskim |
| pubmed-article:12241832 | lifeskim:mentions | umls-concept:C0126174 | lld:lifeskim |
| pubmed-article:12241832 | lifeskim:mentions | umls-concept:C0026565 | lld:lifeskim |
| pubmed-article:12241832 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
| pubmed-article:12241832 | lifeskim:mentions | umls-concept:C0026538 | lld:lifeskim |
| pubmed-article:12241832 | lifeskim:mentions | umls-concept:C0231491 | lld:lifeskim |
| pubmed-article:12241832 | lifeskim:mentions | umls-concept:C2698651 | lld:lifeskim |
| pubmed-article:12241832 | pubmed:issue | 9335 | lld:pubmed |
| pubmed-article:12241832 | pubmed:dateCreated | 2002-9-20 | lld:pubmed |
| pubmed-article:12241832 | pubmed:abstractText | ACE inhibitors attenuate the detrimental effects of angiotensin II, and improve survival and reduce morbidity in patients with acute myocardial infarction and evidence of heart failure or left-ventricular dysfunction. Selective antagonism of the angiotensin type 1 receptor represents an alternative approach to inhibition of the renin-angiotensin system. We did a multicentre, randomised trial to test the hypothesis that the angiotensin II antagonist losartan would be superior or non-inferior to the ACE inhibitor captopril in decreasing all-cause mortality in high-risk patients after acute myocardial infarction. | lld:pubmed |
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| pubmed-article:12241832 | pubmed:language | eng | lld:pubmed |
| pubmed-article:12241832 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:12241832 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:12241832 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:12241832 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:12241832 | pubmed:issn | 0140-6736 | lld:pubmed |
| pubmed-article:12241832 | pubmed:author | pubmed-author:KjekshusJohnJ | lld:pubmed |
| pubmed-article:12241832 | pubmed:author | pubmed-author:DicksteinKenn... | lld:pubmed |
| pubmed-article:12241832 | pubmed:author | pubmed-author:OPTIMAAL... | lld:pubmed |
| pubmed-article:12241832 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:12241832 | pubmed:day | 7 | lld:pubmed |
| pubmed-article:12241832 | pubmed:volume | 360 | lld:pubmed |
| pubmed-article:12241832 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:12241832 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:12241832 | pubmed:pagination | 752-60 | lld:pubmed |
| pubmed-article:12241832 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:12241832 | pubmed:year | 2002 | lld:pubmed |
| pubmed-article:12241832 | pubmed:articleTitle | Effects of losartan and captopril on mortality and morbidity in high-risk patients after acute myocardial infarction: the OPTIMAAL randomised trial. Optimal Trial in Myocardial Infarction with Angiotensin II Antagonist Losartan. | lld:pubmed |
| pubmed-article:12241832 | pubmed:affiliation | University of Bergen, Cardiology Division, Central Hospital in Rogaland, 4011 Stavanger, Norway. trout.@online.no | lld:pubmed |
| pubmed-article:12241832 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:12241832 | pubmed:publicationType | Clinical Trial | lld:pubmed |
| pubmed-article:12241832 | pubmed:publicationType | Randomized Controlled Trial | lld:pubmed |
| pubmed-article:12241832 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:12241832 | pubmed:publicationType | Multicenter Study | lld:pubmed |
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