rdf:type |
|
lifeskim:mentions |
|
pubmed:issue |
7
|
pubmed:dateCreated |
2002-9-18
|
pubmed:abstractText |
Normal bronchial epithelium gradually acquires cellular and genetic changes that result in the formation of invasive tumors. The objective of this study was to evaluate the degree of proliferative change and the amount of neovascularization in both normal and preneoplastic lesions in smokers who were at high risk for developing lung carcinoma.
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pubmed:language |
eng
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pubmed:journal |
|
pubmed:citationSubset |
AIM
|
pubmed:chemical |
|
pubmed:status |
MEDLINE
|
pubmed:month |
Oct
|
pubmed:issn |
0008-543X
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pubmed:author |
|
pubmed:copyrightInfo |
Copyright 2002 American Cancer Society.DOI 10.1002/cncr.10850
|
pubmed:issnType |
Print
|
pubmed:day |
1
|
pubmed:volume |
95
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
1539-45
|
pubmed:dateRevised |
2006-11-15
|
pubmed:meshHeading |
pubmed-meshheading:12237923-Antigens, CD34,
pubmed-meshheading:12237923-Carcinoma, Squamous Cell,
pubmed-meshheading:12237923-Cell Division,
pubmed-meshheading:12237923-Female,
pubmed-meshheading:12237923-Humans,
pubmed-meshheading:12237923-Immunohistochemistry,
pubmed-meshheading:12237923-Ki-67 Antigen,
pubmed-meshheading:12237923-Lung,
pubmed-meshheading:12237923-Lung Neoplasms,
pubmed-meshheading:12237923-Male,
pubmed-meshheading:12237923-Metaplasia,
pubmed-meshheading:12237923-Neovascularization, Pathologic,
pubmed-meshheading:12237923-Precancerous Conditions,
pubmed-meshheading:12237923-Respiratory Mucosa,
pubmed-meshheading:12237923-Smoking,
pubmed-meshheading:12237923-Tumor Suppressor Protein p53
|
pubmed:year |
2002
|
pubmed:articleTitle |
Evidence of neoangiogenesis and an increase in the number of proliferating cells within the bronchial epithelium of smokers.
|
pubmed:affiliation |
Department of Basic Pathology, Graduate School of Medicine, Chiba University, Chiba, Japan. kenzo@med.m.chiba-u.ac.jp
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|