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| pubmed-article:12198705 | lifeskim:mentions | umls-concept:C0542341 | lld:lifeskim |
| pubmed-article:12198705 | lifeskim:mentions | umls-concept:C0442805 | lld:lifeskim |
| pubmed-article:12198705 | lifeskim:mentions | umls-concept:C1333908 | lld:lifeskim |
| pubmed-article:12198705 | lifeskim:mentions | umls-concept:C0031164 | lld:lifeskim |
| pubmed-article:12198705 | lifeskim:mentions | umls-concept:C0179400 | lld:lifeskim |
| pubmed-article:12198705 | lifeskim:mentions | umls-concept:C0872351 | lld:lifeskim |
| pubmed-article:12198705 | lifeskim:mentions | umls-concept:C0173022 | lld:lifeskim |
| pubmed-article:12198705 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:12198705 | pubmed:dateCreated | 2002-8-28 | lld:pubmed |
| pubmed-article:12198705 | pubmed:abstractText | High mobility group (HMG) B1 is a nonhistone nuclear protein that was recently identified as a late-acting mediator of lipopolysaccharide-induced lethality in mice. The proinflammatory actions of HMGB1 have been localized to a region of the molecule called the B box. | lld:pubmed |
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| pubmed-article:12198705 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:12198705 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:12198705 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:12198705 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:12198705 | pubmed:issn | 0016-5085 | lld:pubmed |
| pubmed-article:12198705 | pubmed:author | pubmed-author:FinkMitchell... | lld:pubmed |
| pubmed-article:12198705 | pubmed:author | pubmed-author:YangHuanH | lld:pubmed |
| pubmed-article:12198705 | pubmed:author | pubmed-author:TraceyKevin... | lld:pubmed |
| pubmed-article:12198705 | pubmed:author | pubmed-author:DeludeRussell... | lld:pubmed |
| pubmed-article:12198705 | pubmed:author | pubmed-author:SappingtonPen... | lld:pubmed |
| pubmed-article:12198705 | pubmed:author | pubmed-author:YangRunkuanR | lld:pubmed |
| pubmed-article:12198705 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:12198705 | pubmed:volume | 123 | lld:pubmed |
| pubmed-article:12198705 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:12198705 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:12198705 | pubmed:pagination | 790-802 | lld:pubmed |
| pubmed-article:12198705 | pubmed:dateRevised | 2011-10-27 | lld:pubmed |
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| pubmed-article:12198705 | pubmed:year | 2002 | lld:pubmed |
| pubmed-article:12198705 | pubmed:articleTitle | HMGB1 B box increases the permeability of Caco-2 enterocytic monolayers and impairs intestinal barrier function in mice. | lld:pubmed |
| pubmed-article:12198705 | pubmed:affiliation | Department of Critical Care Medicine, University of Pittsburgh Medical School, Pittsburgh, Pennsylvania 15261, USA. | lld:pubmed |
| pubmed-article:12198705 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:12198705 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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