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rdf:type |
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lifeskim:mentions |
umls-concept:C0009319,
umls-concept:C0011155,
umls-concept:C0017262,
umls-concept:C0026809,
umls-concept:C0031272,
umls-concept:C0162638,
umls-concept:C0185117,
umls-concept:C0205217,
umls-concept:C0332152,
umls-concept:C0332281,
umls-concept:C0376515,
umls-concept:C0392756,
umls-concept:C0684321,
umls-concept:C2003941,
umls-concept:C2911684
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pubmed:issue |
3
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pubmed:dateCreated |
2002-8-12
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pubmed:abstractText |
G protein deficient (G alpha i2-/-) mice spontaneously develop an inflammatory bowel disease (IBD) closely resembling ulcerative colitis. Previous studies have demonstrated that gut T cells are hyperreactive to the endogenous microflora in most IBD models.
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pubmed:commentsCorrections |
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-10029614,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-10432286,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-10467356,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-10483889,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-10528176,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-10549650,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-10790434,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-10848665,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-10886787,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-10917533,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-11049113,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-11101869,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-11286687,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-11368354,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-11413108,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-1534253,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-1681159,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-1833201,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-2139106,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-2507904,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-6607126,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-7663509,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-7751020,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-7768364,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-7964509,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-8370397,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-8402910,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-8536356,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-8684468,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-8760824,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-8770866,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-8945580,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-9013944,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-9207274,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-9353050,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-9453485,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-9453612,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-9454767,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-9771405,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-9818551,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-9842908,
http://linkedlifedata.com/resource/pubmed/commentcorrection/12171962-9918121
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Sep
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pubmed:issn |
0017-5749
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pubmed:author |
|
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pubmed:issnType |
Print
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pubmed:volume |
51
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
392-7
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pubmed:dateRevised |
2009-11-18
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pubmed:meshHeading |
pubmed-meshheading:12171962-Animals,
pubmed-meshheading:12171962-Apoptosis,
pubmed-meshheading:12171962-B-Lymphocytes,
pubmed-meshheading:12171962-Colitis, Ulcerative,
pubmed-meshheading:12171962-Female,
pubmed-meshheading:12171962-GTP-Binding Proteins,
pubmed-meshheading:12171962-Interferon-gamma,
pubmed-meshheading:12171962-Male,
pubmed-meshheading:12171962-Mice,
pubmed-meshheading:12171962-Mice, Inbred Strains,
pubmed-meshheading:12171962-Peyer's Patches,
pubmed-meshheading:12171962-Proto-Oncogene Proteins c-bcl-2,
pubmed-meshheading:12171962-T-Lymphocytes
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pubmed:year |
2002
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pubmed:articleTitle |
Regression of Peyer's patches in G alpha i2 deficient mice prior to colitis is associated with reduced expression of Bcl-2 and increased apoptosis.
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pubmed:affiliation |
Department of Clinical Immunology, Göteborg University, Sweden.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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