Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
4
pubmed:dateCreated
2002-8-8
pubmed:abstractText
Left ventricular (LV) dilation and myocardial remodelling are hallmarks of heart failure in idiopathic dilated cardiomyopathy (DCM). Interstitial collagen is essential for LV integrity and function while degradation of collagen by collagenases, especially matrix-metalloproteinases (MMPs), are suggested to contribute to ventricular dilation. In the present study, serological markers of collagen metabolism were investigated.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Aug
pubmed:issn
1388-9842
pubmed:author
pubmed:issnType
Print
pubmed:volume
4
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
439-4
pubmed:dateRevised
2011-6-8
pubmed:meshHeading
pubmed-meshheading:12167381-Adult, pubmed-meshheading:12167381-Aged, pubmed-meshheading:12167381-Aged, 80 and over, pubmed-meshheading:12167381-Biological Markers, pubmed-meshheading:12167381-Cardiomyopathy, Dilated, pubmed-meshheading:12167381-Collagen, pubmed-meshheading:12167381-Collagenases, pubmed-meshheading:12167381-Female, pubmed-meshheading:12167381-Heart Failure, pubmed-meshheading:12167381-Humans, pubmed-meshheading:12167381-Male, pubmed-meshheading:12167381-Matrix Metalloproteinase 1, pubmed-meshheading:12167381-Matrix Metalloproteinases, pubmed-meshheading:12167381-Middle Aged, pubmed-meshheading:12167381-Myocardium, pubmed-meshheading:12167381-Tissue Inhibitor of Metalloproteinase-1, pubmed-meshheading:12167381-Ventricular Dysfunction, Left, pubmed-meshheading:12167381-Ventricular Remodeling
pubmed:year
2002
pubmed:articleTitle
Elevated serum markers of collagen degradation in patients with mild to moderate dilated cardiomyopathy.
pubmed:affiliation
Department of Cardiology, Angiology and Pneumology, Heinrich-Heine University Düsseldorf, Moorenstrasse 5, Germany. schwartzkopff@med.uni-duesseldorf.de
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't