| pubmed-article:12160190 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:12160190 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
| pubmed-article:12160190 | lifeskim:mentions | umls-concept:C0441833 | lld:lifeskim |
| pubmed-article:12160190 | lifeskim:mentions | umls-concept:C0020538 | lld:lifeskim |
| pubmed-article:12160190 | lifeskim:mentions | umls-concept:C0080103 | lld:lifeskim |
| pubmed-article:12160190 | lifeskim:mentions | umls-concept:C0030190 | lld:lifeskim |
| pubmed-article:12160190 | lifeskim:mentions | umls-concept:C0857121 | lld:lifeskim |
| pubmed-article:12160190 | pubmed:issue | 8 | lld:pubmed |
| pubmed-article:12160190 | pubmed:dateCreated | 2002-8-5 | lld:pubmed |
| pubmed-article:12160190 | pubmed:abstractText | Although the renin-angiotensin system and insulin resistance (IR) have been identified as major regulators of plasminogen activator inhibitor type-1 (PAI-1), their roles in hypertensive subjects is not clearly defined. | lld:pubmed |
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| pubmed-article:12160190 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:12160190 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:12160190 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:12160190 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:12160190 | pubmed:issn | 0895-7061 | lld:pubmed |
| pubmed-article:12160190 | pubmed:author | pubmed-author:BrownNancy... | lld:pubmed |
| pubmed-article:12160190 | pubmed:author | pubmed-author:VaughanDougla... | lld:pubmed |
| pubmed-article:12160190 | pubmed:author | pubmed-author:HopkinsPaul... | lld:pubmed |
| pubmed-article:12160190 | pubmed:author | pubmed-author:JeunemaitreXa... | lld:pubmed |
| pubmed-article:12160190 | pubmed:author | pubmed-author:HuntSteven... | lld:pubmed |
| pubmed-article:12160190 | pubmed:author | pubmed-author:WilliamsGordo... | lld:pubmed |
| pubmed-article:12160190 | pubmed:author | pubmed-author:SrikumarNadar... | lld:pubmed |
| pubmed-article:12160190 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:12160190 | pubmed:volume | 15 | lld:pubmed |
| pubmed-article:12160190 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:12160190 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:12160190 | pubmed:pagination | 683-90 | lld:pubmed |
| pubmed-article:12160190 | pubmed:dateRevised | 2009-2-24 | lld:pubmed |
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| pubmed-article:12160190 | pubmed:meshHeading | pubmed-meshheading:12160190... | lld:pubmed |
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| pubmed-article:12160190 | pubmed:meshHeading | pubmed-meshheading:12160190... | lld:pubmed |
| pubmed-article:12160190 | pubmed:year | 2002 | lld:pubmed |
| pubmed-article:12160190 | pubmed:articleTitle | PAI-1 in human hypertension: relation to hypertensive groups. | lld:pubmed |
| pubmed-article:12160190 | pubmed:affiliation | Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts, USA. | lld:pubmed |
| pubmed-article:12160190 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:12160190 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:12160190 | pubmed:publicationType | Multicenter Study | lld:pubmed |
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| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:12160190 | lld:pubmed |
