Source:http://linkedlifedata.com/resource/pubmed/id/12151853
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
4
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pubmed:dateCreated |
2002-8-1
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pubmed:abstractText |
Despite changes in lifestyle and the use of effective pharmacologic interventions to lower cholesterol levels, coronary heart disease remains the major cause of morbidity and mortality in the developed world. Cholesterol screening fails to identify almost 50% of those individuals who will present with acute coronary syndromes. Recent evidence from laboratory and prospective clinical studies demonstrates that atherosclerosis is not simply a disease of lipid deposition, but rather is an inflammatory process with highly specific cellular and molecular responses. The clinical utility of inflammatory markers has been examined in a variety of atherothrombotic diseases. Because C-reactive protein is highly stable in stored frozen samples, and automated and robust analytical systems for its measurement are available, it has become the most widely examined inflammatory marker.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Aug
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pubmed:issn |
0957-9672
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
13
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
383-9
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pubmed:dateRevised |
2005-11-16
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pubmed:meshHeading | |
pubmed:year |
2002
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pubmed:articleTitle |
Inflammatory markers and coronary heart disease.
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pubmed:affiliation |
Department of Laboratory Medicine, Children's Hospital, Boston, Massachusetts 02115, USA. nader.rifai@tch.harvard.edu
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pubmed:publicationType |
Journal Article,
Review
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