Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
1
pubmed:dateCreated
2002-7-22
pubmed:abstractText
Ischemia/reperfusion (I/R) injury is a critical factor in the dysfunction of steatotic orthotopic liver transplants. Heme oxygenase-1 (HO-1), a cytoprotective protein, may be important in ameliorating hepatic I/R injury.
pubmed:grant
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Jul
pubmed:issn
0041-1337
pubmed:author
pubmed:issnType
Print
pubmed:day
15
pubmed:volume
74
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
96-102
pubmed:dateRevised
2007-11-14
pubmed:meshHeading
pubmed-meshheading:12134106-Animals, pubmed-meshheading:12134106-Apoptosis, pubmed-meshheading:12134106-Gene Expression Regulation, Enzymologic, pubmed-meshheading:12134106-Gene Therapy, pubmed-meshheading:12134106-Graft Survival, pubmed-meshheading:12134106-Heme Oxygenase (Decyclizing), pubmed-meshheading:12134106-Heme Oxygenase-1, pubmed-meshheading:12134106-Liver, pubmed-meshheading:12134106-Liver Transplantation, pubmed-meshheading:12134106-Macrophages, pubmed-meshheading:12134106-Male, pubmed-meshheading:12134106-Neutrophils, pubmed-meshheading:12134106-Nitric Oxide Synthase, pubmed-meshheading:12134106-Nitric Oxide Synthase Type II, pubmed-meshheading:12134106-Nitric Oxide Synthase Type III, pubmed-meshheading:12134106-Obesity, pubmed-meshheading:12134106-Rats, pubmed-meshheading:12134106-Rats, Zucker, pubmed-meshheading:12134106-Reperfusion Injury, pubmed-meshheading:12134106-T-Lymphocytes, pubmed-meshheading:12134106-Transfection
pubmed:year
2002
pubmed:articleTitle
Heme oxygenase-1 gene transfer inhibits inducible nitric oxide synthase expression and protects genetically fat Zucker rat livers from ischemia-reperfusion injury.
pubmed:affiliation
Dumont-UCLA Transplant Center, Department of Surgery, UCLA School of Medicine, 90095, USA.
pubmed:publicationType
Journal Article, Research Support, U.S. Gov't, P.H.S., Research Support, Non-U.S. Gov't