Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
6
pubmed:dateCreated
2002-7-10
pubmed:abstractText
This study examined whether locally expressed CTLA4-Ig can suppress the accelerated islet allograft rejection that is induced by donor-specific transfusion.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Jun
pubmed:issn
0012-186X
pubmed:author
pubmed:issnType
Print
pubmed:volume
45
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
831-40
pubmed:dateRevised
2011-11-17
pubmed:meshHeading
pubmed-meshheading:12107727-Animals, pubmed-meshheading:12107727-Antigens, CD, pubmed-meshheading:12107727-Antigens, Differentiation, pubmed-meshheading:12107727-Blood Glucose, pubmed-meshheading:12107727-COS Cells, pubmed-meshheading:12107727-CTLA-4 Antigen, pubmed-meshheading:12107727-Cell Line, pubmed-meshheading:12107727-Cercopithecus aethiops, pubmed-meshheading:12107727-Diabetes Mellitus, Experimental, pubmed-meshheading:12107727-Graft Rejection, pubmed-meshheading:12107727-Immunoconjugates, pubmed-meshheading:12107727-Immunoglobulin Fc Fragments, pubmed-meshheading:12107727-Insulinoma, pubmed-meshheading:12107727-Islets of Langerhans, pubmed-meshheading:12107727-Islets of Langerhans Transplantation, pubmed-meshheading:12107727-Lymphocyte Transfusion, pubmed-meshheading:12107727-Male, pubmed-meshheading:12107727-Mice, pubmed-meshheading:12107727-Mice, Inbred C3H, pubmed-meshheading:12107727-Mice, Inbred C57BL, pubmed-meshheading:12107727-Pancreatic Neoplasms, pubmed-meshheading:12107727-Recombinant Proteins, pubmed-meshheading:12107727-Spleen, pubmed-meshheading:12107727-Transfection, pubmed-meshheading:12107727-Transplantation, Homologous, pubmed-meshheading:12107727-Tumor Cells, Cultured
pubmed:year
2002
pubmed:articleTitle
Locally expressed CTLA4-Ig in a pancreatic beta-cell line suppresses accelerated graft rejection response induced by donor-specific transfusion.
pubmed:affiliation
Molecular and Cellular Recognition, Department of Surgery and Clinical Oncology, Osaka University Graduate School of Medicine, Osaka, Japan.
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't