Linked Life Data

12080443

Source:http://linkedlifedata.com/resource/pubmed/id/12080443

Statements in which the resource exists.
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pubmed-article:12080443pubmed:abstractTextLeptin, a hormone produced in adipocytes, is a key signal in the regulation of food intake and energy expenditure. Beta-adrenergic agonists have been shown to inhibit leptin gene expression and leptin secretion. The mechanisms underlying the inhibitory effects of beta-adrenergic agonists have not been established. In this study, we examined the effects of Trecadrine, a novel beta3-adrenergic agonist, on basal and insulin-stimulated leptin secretion in isolated rat adipocytes. Because insulin-stimulated glucose metabolism is an important regulator of leptin expression and secretion by the adipocytes, the effects of Trecadrine on indices of adipocyte metabolism were also examined.lld:pubmed
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pubmed-article:12080443pubmed:authorpubmed-author:MartínezJ AJAlld:pubmed
pubmed-article:12080443pubmed:authorpubmed-author:HavelP JPJlld:pubmed
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pubmed-article:12080443pubmed:pagination912-9lld:pubmed
pubmed-article:12080443pubmed:dateRevised2007-11-14lld:pubmed
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pubmed-article:12080443pubmed:year2002lld:pubmed
pubmed-article:12080443pubmed:articleTitleEffects of Trecadrine, a beta3-adrenergic agonist, on leptin secretion, glucose and lipid metabolism in isolated rat adipocytes.lld:pubmed
pubmed-article:12080443pubmed:affiliationDepartment of Physiology and Nutrition, University of Navarra, Pamplona, Spain.lld:pubmed
pubmed-article:12080443pubmed:publicationTypeJournal Articlelld:pubmed
pubmed-article:12080443pubmed:publicationTypeResearch Support, U.S. Gov't, P.H.S.lld:pubmed
pubmed-article:12080443pubmed:publicationTypeResearch Support, U.S. Gov't, Non-P.H.S.lld:pubmed
pubmed-article:12080443pubmed:publicationTypeResearch Support, Non-U.S. Gov'tlld:pubmed