rdf:type |
|
lifeskim:mentions |
umls-concept:C0003483,
umls-concept:C0017262,
umls-concept:C0034693,
umls-concept:C0034721,
umls-concept:C0185117,
umls-concept:C0205245,
umls-concept:C0392747,
umls-concept:C0439799,
umls-concept:C0443172,
umls-concept:C0458003,
umls-concept:C0521116,
umls-concept:C0596981,
umls-concept:C0678723,
umls-concept:C1521970,
umls-concept:C2911684
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pubmed:issue |
1
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pubmed:dateCreated |
2002-6-13
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pubmed:abstractText |
Active control of the arterial diameter by vascular smooth muscle is one of the principle mechanisms by which vessels adapt to a significant rise in blood pressure after birth. Although voltage-gated K+ (Kv) channels play an important role in the regulation of excitation-contraction coupling in arteries, very little is known about postnatal modification of Kv channels. We therefore investigated changes in the functional characteristics and expression of Kv channels in rat aortic myocytes (RAMs) during early postnatal development.
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pubmed:language |
eng
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pubmed:journal |
|
pubmed:citationSubset |
IM
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pubmed:chemical |
http://linkedlifedata.com/resource/pubmed/chemical/4-Aminopyridine,
http://linkedlifedata.com/resource/pubmed/chemical/Delayed Rectifier Potassium Channels,
http://linkedlifedata.com/resource/pubmed/chemical/Kcna2 protein, rat,
http://linkedlifedata.com/resource/pubmed/chemical/Kcna5 protein, rat,
http://linkedlifedata.com/resource/pubmed/chemical/Kcnb1 protein, rat,
http://linkedlifedata.com/resource/pubmed/chemical/Kv1.2 Potassium Channel,
http://linkedlifedata.com/resource/pubmed/chemical/Kv1.5 Potassium Channel,
http://linkedlifedata.com/resource/pubmed/chemical/Potassium Channel Blockers,
http://linkedlifedata.com/resource/pubmed/chemical/Potassium Channels,
http://linkedlifedata.com/resource/pubmed/chemical/Potassium Channels, Voltage-Gated,
http://linkedlifedata.com/resource/pubmed/chemical/Shab Potassium Channels,
http://linkedlifedata.com/resource/pubmed/chemical/Tetraethylammonium
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pubmed:status |
MEDLINE
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pubmed:month |
Apr
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pubmed:issn |
0008-6363
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pubmed:author |
|
pubmed:issnType |
Print
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pubmed:volume |
54
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
152-61
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:12062371-4-Aminopyridine,
pubmed-meshheading:12062371-Action Potentials,
pubmed-meshheading:12062371-Animals,
pubmed-meshheading:12062371-Animals, Newborn,
pubmed-meshheading:12062371-Aorta,
pubmed-meshheading:12062371-Blotting, Western,
pubmed-meshheading:12062371-Delayed Rectifier Potassium Channels,
pubmed-meshheading:12062371-Kv1.2 Potassium Channel,
pubmed-meshheading:12062371-Kv1.5 Potassium Channel,
pubmed-meshheading:12062371-Male,
pubmed-meshheading:12062371-Muscle, Smooth, Vascular,
pubmed-meshheading:12062371-Patch-Clamp Techniques,
pubmed-meshheading:12062371-Potassium Channel Blockers,
pubmed-meshheading:12062371-Potassium Channels,
pubmed-meshheading:12062371-Potassium Channels, Voltage-Gated,
pubmed-meshheading:12062371-Rats,
pubmed-meshheading:12062371-Rats, Wistar,
pubmed-meshheading:12062371-Shab Potassium Channels,
pubmed-meshheading:12062371-Tetraethylammonium
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pubmed:year |
2002
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pubmed:articleTitle |
Developmental changes in the functional characteristics and expression of voltage-gated K+ channel currents in rat aortic myocytes.
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pubmed:affiliation |
Centre for Cardiovascular Biology and Medicine, King's College London, St Thomas's Campus, SE1 7EH, UK.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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