| pubmed-article:12050398 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:12050398 | lifeskim:mentions | umls-concept:C0013480 | lld:lifeskim |
| pubmed-article:12050398 | lifeskim:mentions | umls-concept:C0913092 | lld:lifeskim |
| pubmed-article:12050398 | lifeskim:mentions | umls-concept:C0178539 | lld:lifeskim |
| pubmed-article:12050398 | lifeskim:mentions | umls-concept:C0907411 | lld:lifeskim |
| pubmed-article:12050398 | lifeskim:mentions | umls-concept:C0086597 | lld:lifeskim |
| pubmed-article:12050398 | lifeskim:mentions | umls-concept:C1550548 | lld:lifeskim |
| pubmed-article:12050398 | lifeskim:mentions | umls-concept:C1555714 | lld:lifeskim |
| pubmed-article:12050398 | lifeskim:mentions | umls-concept:C1705654 | lld:lifeskim |
| pubmed-article:12050398 | lifeskim:mentions | umls-concept:C1504308 | lld:lifeskim |
| pubmed-article:12050398 | pubmed:issue | 13 | lld:pubmed |
| pubmed-article:12050398 | pubmed:dateCreated | 2002-6-6 | lld:pubmed |
| pubmed-article:12050398 | pubmed:abstractText | Ebola virus is a highly lethal pathogen responsible for several outbreaks of hemorrhagic fever. Here we show that the primate lentiviral binding C-type lectins DC-SIGN and L-SIGN act as cofactors for cellular entry by Ebola virus. Furthermore, DC-SIGN on the surface of dendritic cells is able to function as a trans receptor, binding Ebola virus-pseudotyped lentiviral particles and transmitting infection to susceptible cells. Our data underscore a role for DC-SIGN and L-SIGN in the infective process and pathogenicity of Ebola virus infection. | lld:pubmed |
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| pubmed-article:12050398 | pubmed:language | eng | lld:pubmed |
| pubmed-article:12050398 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:12050398 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:12050398 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:12050398 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:12050398 | pubmed:issn | 0022-538X | lld:pubmed |
| pubmed-article:12050398 | pubmed:author | pubmed-author:DelgadoRafael... | lld:pubmed |
| pubmed-article:12050398 | pubmed:author | pubmed-author:LasalaFátimaF | lld:pubmed |
| pubmed-article:12050398 | pubmed:author | pubmed-author:AlvarezCarmen... | lld:pubmed |
| pubmed-article:12050398 | pubmed:author | pubmed-author:CorbíAngel... | lld:pubmed |
| pubmed-article:12050398 | pubmed:author | pubmed-author:CarrilloJaime... | lld:pubmed |
| pubmed-article:12050398 | pubmed:author | pubmed-author:MuñizOscarO | lld:pubmed |
| pubmed-article:12050398 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:12050398 | pubmed:volume | 76 | lld:pubmed |
| pubmed-article:12050398 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:12050398 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:12050398 | pubmed:pagination | 6841-4 | lld:pubmed |
| pubmed-article:12050398 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:12050398 | pubmed:year | 2002 | lld:pubmed |
| pubmed-article:12050398 | pubmed:articleTitle | C-type lectins DC-SIGN and L-SIGN mediate cellular entry by Ebola virus in cis and in trans. | lld:pubmed |
| pubmed-article:12050398 | pubmed:affiliation | Laboratory of Molecular Microbiology, Dept. of Microbiology, Hospital 12 de Octubre, 28041 Madrid, Spain. | lld:pubmed |
| pubmed-article:12050398 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:12050398 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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