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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
6
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pubmed:dateCreated |
2002-6-5
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pubmed:abstractText |
Prominent infiltration of eosinophils in airway mucosa is the pathognomonic sign of asthma. The role of airway epithelial cells in eosinophil infiltration, however, has not been fully elucidated.
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pubmed:commentsCorrections |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
|
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pubmed:status |
MEDLINE
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pubmed:month |
Jun
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pubmed:issn |
0954-7894
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
32
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
889-97
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:12047436-Bronchi,
pubmed-meshheading:12047436-Cell Movement,
pubmed-meshheading:12047436-Chemokines,
pubmed-meshheading:12047436-Chemotaxis, Leukocyte,
pubmed-meshheading:12047436-Collagen Type IV,
pubmed-meshheading:12047436-Eosinophils,
pubmed-meshheading:12047436-Epithelial Cells,
pubmed-meshheading:12047436-Humans,
pubmed-meshheading:12047436-Interleukin-4,
pubmed-meshheading:12047436-Neutrophils,
pubmed-meshheading:12047436-Respiratory Mucosa,
pubmed-meshheading:12047436-Tumor Necrosis Factor-alpha
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pubmed:year |
2002
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pubmed:articleTitle |
Airway epithelial cells promote transmigration of eosinophils in a new three-dimensional chemotaxis model.
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pubmed:affiliation |
Department of Psychiatry, University of Mie School of Medicine, Tsu, Japan.
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pubmed:publicationType |
Journal Article,
Comparative Study,
Research Support, Non-U.S. Gov't
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