Source:http://linkedlifedata.com/resource/pubmed/id/11999585
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
2
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pubmed:dateCreated |
2002-5-9
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pubmed:abstractText |
The development of a IgGk monoclonal gammopathy after a phase of bone marrow aplasia following chemotherapy is described in a patient suffering from biphenotypic acute leukemia (BAL). Paraprotein was followed by the relapse of the disease and disappeared during a further chemotherapy. Paraprotein could have been caused by an additional chemotherapy-induced genetic mutation or by a dysfunction in T-B cooperation observed in the phase of reconstitution of the immune system after medullar aplasia.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:status |
MEDLINE
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pubmed:month |
Feb
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pubmed:issn |
1042-8194
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
43
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
441-3
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pubmed:dateRevised |
2006-4-24
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pubmed:meshHeading |
pubmed-meshheading:11999585-Acute Disease,
pubmed-meshheading:11999585-Adult,
pubmed-meshheading:11999585-Antineoplastic Combined Chemotherapy Protocols,
pubmed-meshheading:11999585-Clone Cells,
pubmed-meshheading:11999585-Female,
pubmed-meshheading:11999585-Humans,
pubmed-meshheading:11999585-Immunophenotyping,
pubmed-meshheading:11999585-Karyotyping,
pubmed-meshheading:11999585-Leukemia,
pubmed-meshheading:11999585-Paraproteinemias,
pubmed-meshheading:11999585-Phenotype
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pubmed:year |
2002
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pubmed:articleTitle |
Development of a transient monoclonal gammopathy after chemotherapy in a patient with biphenotypic acute leukemia.
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pubmed:affiliation |
Department of Haematology, Transfusion Medicine and Transplant Immunology, Second University of Naples, Italy. roberto.guariglia@uninaz.it
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pubmed:publicationType |
Journal Article,
Case Reports
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