rdf:type |
|
lifeskim:mentions |
|
pubmed:issue |
3
|
pubmed:dateCreated |
2002-4-16
|
pubmed:abstractText |
To confirm the role of HLA-B2704 and hbeta(2)m gene in the pathogenesis of spontaneous inflammatory diseases by establishing HLA-B2704 and hbeta(2)m double transgenic mice model of ankylosing spondylitis. It will provide a powerful animal model for exploring the etiology, prevention and treatment of B27-relevant diseases.
|
pubmed:language |
chi
|
pubmed:journal |
|
pubmed:citationSubset |
IM
|
pubmed:chemical |
|
pubmed:status |
MEDLINE
|
pubmed:month |
Mar
|
pubmed:issn |
0529-5815
|
pubmed:author |
|
pubmed:issnType |
Print
|
pubmed:volume |
40
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
216-8
|
pubmed:dateRevised |
2006-11-15
|
pubmed:meshHeading |
pubmed-meshheading:11955421-Animals,
pubmed-meshheading:11955421-Disease Models, Animal,
pubmed-meshheading:11955421-HLA-B27 Antigen,
pubmed-meshheading:11955421-Inflammation,
pubmed-meshheading:11955421-Mice,
pubmed-meshheading:11955421-Mice, Transgenic,
pubmed-meshheading:11955421-Reverse Transcriptase Polymerase Chain Reaction,
pubmed-meshheading:11955421-Spondylitis, Ankylosing
|
pubmed:year |
2002
|
pubmed:articleTitle |
[Spontaneous inflammatory diseases in ankylosing spondylitis transgenic mice].
|
pubmed:affiliation |
Arthritis Clinical and Research Center, People's Hospital, Peking University, Beijing 100044, China.
|
pubmed:publicationType |
Journal Article,
English Abstract
|