rdf:type |
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lifeskim:mentions |
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pubmed:issue |
6
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pubmed:dateCreated |
2002-1-23
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pubmed:abstractText |
PURPOSE/AIMS: The prenatal estrogen excess hypothesis postulates abnormally high estrogen levels during pregnancy which predispose the developing gonad to testicular germ cell cancer (GCT) in adulthood. As no direct measurements are possible to support this hypothesis, evidence must come from clinical and epidemiological observations. The present study looked to surrogate parameters that purportedly point to high estrogenic influence in utero.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Dec
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pubmed:issn |
0302-2838
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pubmed:author |
|
pubmed:issnType |
Print
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pubmed:volume |
40
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
677-83; discussion 684
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pubmed:dateRevised |
2011-1-11
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pubmed:meshHeading |
pubmed-meshheading:11805417-Adult,
pubmed-meshheading:11805417-Birth Order,
pubmed-meshheading:11805417-Breast Neoplasms,
pubmed-meshheading:11805417-Case-Control Studies,
pubmed-meshheading:11805417-Estrogens,
pubmed-meshheading:11805417-Female,
pubmed-meshheading:11805417-Germinoma,
pubmed-meshheading:11805417-Humans,
pubmed-meshheading:11805417-Male,
pubmed-meshheading:11805417-Maternal Age,
pubmed-meshheading:11805417-Middle Aged,
pubmed-meshheading:11805417-Paternal Age,
pubmed-meshheading:11805417-Pregnancy,
pubmed-meshheading:11805417-Prenatal Exposure Delayed Effects,
pubmed-meshheading:11805417-Risk Factors,
pubmed-meshheading:11805417-Testicular Neoplasms,
pubmed-meshheading:11805417-Twins
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pubmed:year |
2001
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pubmed:articleTitle |
How valid is the prenatal estrogen excess hypothesis of testicular germ cell cancer? A case control study on hormone-related factors.
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pubmed:affiliation |
Department of Urology, Albertinen-Krankenhaus Hamburg, Germany. DieckmannKP@t-online.de
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pubmed:publicationType |
Journal Article
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