Source:http://linkedlifedata.com/resource/pubmed/id/11797997
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
12
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pubmed:dateCreated |
2002-1-18
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pubmed:abstractText |
It is known that high-strength shock disrupts the lipid matrix of the myocardial cell membrane and forms reversible aqueous pores across the membrane. This process is known as "electroporation." However, it remains unclear whether electroporation contributes to the mechanism of ventricular defibrillation. The aim of this computer simulation study was to examine the possible role of electroporation in the success of defibrillation shock.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:status |
MEDLINE
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pubmed:month |
Dec
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pubmed:issn |
1045-3873
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
12
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1393-403
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pubmed:dateRevised |
2007-11-15
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pubmed:meshHeading |
pubmed-meshheading:11797997-Action Potentials,
pubmed-meshheading:11797997-Anisotropy,
pubmed-meshheading:11797997-Arrhythmias, Cardiac,
pubmed-meshheading:11797997-Computer Simulation,
pubmed-meshheading:11797997-Electric Countershock,
pubmed-meshheading:11797997-Electroporation,
pubmed-meshheading:11797997-Humans,
pubmed-meshheading:11797997-Membrane Potentials,
pubmed-meshheading:11797997-Models, Cardiovascular,
pubmed-meshheading:11797997-Myocardium
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pubmed:year |
2001
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pubmed:articleTitle |
Electroporation in a model of cardiac defibrillation.
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pubmed:affiliation |
First Department of Internal Medicine, Shiga University of Medical Science, Otsu, Japan. ash@belle.shiga-med.ac.jp
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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